Ekg module 6


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Ekg module 6

  1. 1. ECG Rhythm Interpretation Module VIAdvanced 12-Lead Interpretation
  2. 2. Course Objectives• To recognize the normal rhythm of the heart - “Normal Sinus Rhythm.”• To recognize the 13 most common heart arrhythmias.• To recognize an acute myocardial infarction on a 12-lead ECG.
  3. 3. Learning Modules• ECG Basics• How to Analyze a Rhythm• Normal Sinus Rhythm• Heart Arrhythmias• Diagnosing a Myocardial Infarction• Advanced 12-Lead Interpretation
  4. 4. The 12-Lead ECGThe 12-Lead ECG contains a wealth ofinformation. In Module V you learned thatST segment elevation in two leads issuggestive of an acute myocardialinfarction. In this module we will cover: – ST Elevation and non-ST Elevation MIs – Left Ventricular Hypertrophy – Bundle Branch Blocks
  5. 5. ST Elevation andnon-ST Elevation MIs
  6. 6. ST Elevation and non-ST Elevation MIs• When myocardial blood supply is abruptly reduced or cut off to a region of the heart, a sequence of injurious events occur beginning with ischemia (inadequate tissue perfusion), followed by necrosis (infarction), and eventual fibrosis (scarring) if the blood supply isnt restored in an appropriate period of time.• The ECG changes over time with each of these events…
  7. 7. ECG ChangesWays the ECG can change include: ST elevation & depression T-waves peaked flattened invertedAppearanceof pathologicQ-waves
  8. 8. ECG Changes & the Evolving MI Non-ST ElevationThere are twodistinct patternsof ECG changedepending if theinfarction is: ST Elevation–ST Elevation (Transmural or Q-wave), or–Non-ST Elevation (Subendocardial or non-Q-wave)
  9. 9. ST Elevation InfarctionThe ECG changes seen with a ST elevation infarction are: Before injury Normal ECG Ischemia ST depression, peaked T-waves, then T-wave inversion Infarction ST elevation & appearance of Q-waves Fibrosis ST segments and T-waves return to normal, but Q-waves persist
  10. 10. ST Elevation InfarctionHere’s a diagram depicting an evolving infarction:A. Normal ECG prior to MIB. Ischemia from coronary artery occlusion results in ST depression (not shown) and peaked T-wavesC. Infarction from ongoing ischemia results in marked ST elevationD/E. Ongoing infarction with appearance of pathologic Q-waves and T-wave inversionF. Fibrosis (months later) with persistent Q- waves, but normal ST segment and T- waves
  11. 11. ST Elevation InfarctionHere’s an ECG of an inferior MI:Look at theinferior leads(II, III, aVF).Question:What ECGchanges doyou see?ST elevationand Q-wavesExtra credit:What is therhythm? Atrial fibrillation (irregularly irregular with narrow QRS)!
  12. 12. Non-ST Elevation InfarctionHere’s an ECG of an inferior MI later in time:Now what doyou see in theinferior leads?ST elevation,Q-waves andT-waveinversion
  13. 13. Non-ST Elevation InfarctionThe ECG changes seen with a non-ST elevation infarction are:Before injury Normal ECGIschemia ST depression & T-wave inversionInfarction ST depression & T-wave inversionFibrosis ST returns to baseline, but T-wave inversion persists
  14. 14. Non-ST Elevation InfarctionHere’s an ECG of an evolving non-ST elevation MI:Note the STdepressionand T-waveinversion inleads V2-V6.Question:What area ofthe heart isinfarcting?Anterolateral
  15. 15. Left Ventricular Hypertrophy
  16. 16. Left Ventricular Hypertrophy Compare these two 12-lead ECGs. What stands out as different with the second one? Normal Left Ventricular HypertrophyAnswer: The QRS complexes are very tall (increased voltage)
  17. 17. Left Ventricular HypertrophyWhy is left ventricular hypertrophy characterized by tallQRS complexes? As the heart muscle wall thickens there is an increase in electrical forces moving through the myocardium resulting in increased QRS voltage. LVH ECHOcardiogram Increased QRS voltage
  18. 18. Left Ventricular Hypertrophy• Criteria exists to diagnose LVH using a 12-lead ECG. – For example: • The R wave in V5 or V6 plus the S wave in V1 or V2 exceeds 35 mm.• However, for now, all you need to know is that the QRS voltage increases with LVH.
  19. 19. Bundle Branch Blocks
  20. 20. Bundle Branch BlocksTurning our attention to bundle branch blocks… Remember normal impulse conduction is SA node  AV node  Bundle of His  Bundle Branches  Purkinje fibers
  21. 21. Normal Impulse ConductionSinoatrial node AV node Bundle of HisBundle Branches Purkinje fibers
  22. 22. Bundle Branch BlocksSo, depolarization ofthe Bundle Branchesand Purkinje fibers areseen as the QRScomplex on the ECG.Therefore, a conductionblock of the BundleBranches would be Rightreflected as a change in BBBthe QRS complex.
  23. 23. Bundle Branch BlocksWith Bundle Branch Blocks you will see two changeson the ECG. 1. QRS complex widens (> 0.12 sec). 2. QRS morphology changes (varies depending on ECG lead, and if it is a right vs. left bundle branch block).
  24. 24. Bundle Branch BlocksWhy does the QRS complex widen?When the conductionpathway is blocked itwill take longer forthe electrical signalto pass throughoutthe ventricles.
  25. 25. Right Bundle Branch BlocksWhat QRS morphology is characteristic?For RBBB the wide QRS complex assumes aunique, virtually diagnostic shape in thoseleads overlying the right ventricle (V1 and V2). V1 “Rabbit Ears”
  26. 26. Left Bundle Branch BlocksWhat QRS morphology is characteristic?For LBBB the wide QRS complex assumes acharacteristic change in shape in those leadsopposite the left ventricle (right ventricularleads - V1 and V2). Broad,Normal deep S waves
  27. 27. SummaryThis Module introduced you to: – ST Elevation and Non-ST Elevation MIs – Left Ventricular Hypertrophy – Bundle Branch BlocksDon’t worry too much right now about trying toremember all the details. You’ll focus more onadvanced ECG interpretation in your clinicalyears!