NSA MV Oyster 2008

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NSA MV Oyster 2008

  1. 1. Characterizing disease resistance in native oysters that have experienced disease pressure Steven Roberts, Roxanna Smolowitz, Rick Karney Northeastern Regional Aquaculture Center
  2. 2. Disease MVineyard ? Resistance flickr | cpurrin1 Roxannna Smolowitz Rick Karney
  3. 3. Market size
  4. 4. Without Pressure apoptosis protease
  5. 5. n.d.
  6. 6. n.d.
  7. 7. High Mobility Group Protein
  8. 8. Apoptosis – BCL-X • Resistant oyster strains could downregulate apoptosis suppression • Allowing for increased apoptosis • Decreasing number of cells available for Perkinsus proliferation
  9. 9. Schematic Parasite Hemocytes Text BCL-X BCL-X Resistant Wild-type
  10. 10. causative or associative?
  11. 11. Does it matter? Markers ?
  12. 12. Summary • Offspring of survivors of heavy disease pressure are more tolerant to disease • Mechanisms involved in host responses to P. marinus include proteases, apoptosis, and molecule signaling • Careful selection of broodstock (potentially aided by markers) can benefit aquaculture and conservation efforts
  13. 13. Future • Increased analysis on current data set • Analyze gene expression patterns in different locations under different diseased pressure • Application of markers • Could assignment be made via gene expression?

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