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NSA MV Oyster 2008
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NSA MV Oyster 2008

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Transcript

  • 1. Characterizing disease resistance in native oysters that have experienced disease pressure Steven Roberts, Roxanna Smolowitz, Rick Karney Northeastern Regional Aquaculture Center
  • 2. Disease MVineyard ? Resistance flickr | cpurrin1 Roxannna Smolowitz Rick Karney
  • 3. Market size
  • 4. Without Pressure apoptosis protease
  • 5. n.d.
  • 6. n.d.
  • 7. High Mobility Group Protein
  • 8. Apoptosis – BCL-X • Resistant oyster strains could downregulate apoptosis suppression • Allowing for increased apoptosis • Decreasing number of cells available for Perkinsus proliferation
  • 9. Schematic Parasite Hemocytes Text BCL-X BCL-X Resistant Wild-type
  • 10. causative or associative?
  • 11. Does it matter? Markers ?
  • 12. Summary • Offspring of survivors of heavy disease pressure are more tolerant to disease • Mechanisms involved in host responses to P. marinus include proteases, apoptosis, and molecule signaling • Careful selection of broodstock (potentially aided by markers) can benefit aquaculture and conservation efforts
  • 13. Future • Increased analysis on current data set • Analyze gene expression patterns in different locations under different diseased pressure • Application of markers • Could assignment be made via gene expression?