• Share
  • Email
  • Embed
  • Like
  • Save
  • Private Content
Paediatric Cardiology for General Paediatricians presentation
 

Paediatric Cardiology for General Paediatricians presentation

on

  • 1,032 views

 

Statistics

Views

Total Views
1,032
Views on SlideShare
1,032
Embed Views
0

Actions

Likes
0
Downloads
42
Comments
0

0 Embeds 0

No embeds

Accessibility

Categories

Upload Details

Uploaded via as Microsoft PowerPoint

Usage Rights

© All Rights Reserved

Report content

Flagged as inappropriate Flag as inappropriate
Flag as inappropriate

Select your reason for flagging this presentation as inappropriate.

Cancel
  • Full Name Full Name Comment goes here.
    Are you sure you want to
    Your message goes here
    Processing…
Post Comment
Edit your comment

    Paediatric Cardiology for General Paediatricians presentation Paediatric Cardiology for General Paediatricians presentation Presentation Transcript

    • Paediatric Cardiology for General Paediatricians Dr Talal Farha Consultant Paediatrician SpR Regional Teaching Taunton 22 Jan 2008
    • Essentials in looking at an ECG Rhythm (sinus….nonsinus) Rate, Atrial and ventricular rates. QRS axis, T axis, QRS-T angle Intervals: PR. QRS, and QT P wave amplitude and duration QRS amplitude and R/S ratio Q wave St- Segment and T wave abnormalities
    • ECG tips
      • How do you determine Sinus rhythm?
      • What is T axis?
      • What is QRS/T angle?
    • Rhythm
      • P before every QRS
      • P axis (0-90). P inverted in aVR
    • P wave axis
      • The location of the P-wave axis determines the origin of an atrial-derived rhythm:
            • 0 to 90 degrees = a high right (normal sinus rhythm)
            • 90 to 180 degrees = a high left
            • 180 to 270 degrees = a low left
            • 270 to 0 degrees = a low right
    • T wave
      • In most leads, the T wave is positive.
      • A negative T wave is normal in lead aVR.
      • Lead V1 may have a positive, negative, or biphasic T wave. In addition
      • It is not uncommon to have an isolated negative T wave in lead III, aVL, or aVF.
      • Inverted (or negative) T waves can be a sign of
      • Coronary ischemia
      • Left ventricular hypertrophy
    • T axis
      • Determined by the same methods as QRS
      • 0 to + 90 is normal
      • T Axis out side the normal quadrant could suggest conditions with Myocardial dysfunction.
    • QRS-T Angle
      • Formed by the QRS axis and the T axis
      • QRS-T angle >60 degrees is unusual but if > 90 degrees, it is abnormal.
      • Abnormally wide angle, with T axis outside the normal quadrant is seen in
      • - severe ventricular hypertrophy with starin
      • - Ventricular conduction disturbances
      • - Myocardial dysfunction of a metabolic or ischemic nature.
    •  
    • Top Tip For ECG
      • Read more ECGs
    • Do not forget, nothing replaces good traditional clinical examination and detailed history
    • Syncope
      • How often related to the heart?
      • What are the related cardiac conditions?
      • How do we approach it?
    • Definition
      • Syncope is a transient loss of consciousness and muscle tone.
      • Near syncope:
      • premonitory signs and symptoms of imminent syncope occur; dizziness with or without blackout, pallor, diaphoresis, thready pulse and low BP
    • Cause
      • Brain function depends on Oxygen and glucose.
      • Circulatory, metabolic, or neuropsychiatric causes.
      • Adults syncope mostly cardiac.
      • Children’s mostly benign.
    • Causes of Syncope in Children
      • Extra cardiac causes
            • Vasovagal
            • Orthostatic
            • Failure of systemic venous return
            • Cerebrovascular occlusive disease
            • Hyperventilation
            • Breath holding
    • 1- Vasovagal Syncope Neurocardiogenic Common Syncope
      • Predrome for few seconds; dizziness, light-headedness, pallor, palpitation, nausea, hyperventilation then Loss of consciousness and muscle tone
      • Falls without injury
      • Lasts about a minute, awake gradually
    • Vasovagal Syncope
      • Anxiety
      • Fright
      • Pain
      • Blood
      • Fasting
      • Hot and humid conditions
      • Crowded places
      • Prolonged motionless standing
    • Vasovagal Syncope Pathophysiology
      • Standing posture without movement shifts blood to the lower extremities
      • Decrease venous return, stroke volume, BP
      • Less stretching of vent muscle and mechanoreceptors (mrcpts), decline in neural traffic form mrcpts, decreased arterial pressure, increase sympathetic output with
      • Higher HR, vasoconstriction (higher diastolic pressure)
    • Vasovagal Syncope Patients
      • Decreased venous return produces large increase in ventricular contraction force
      • Activation of LV mechanoreceptors (normally only responds to stretch)
      • Increase neural traffic mimicking high BP condition
      • Paradoxical withdrawal of sympathetic activity, vasodilatation, hypotension and bradycardia
      • Reduction of brain perfusion
    • Diagnoses
      • ECG, Holter, EEG, glucose tolerance test all are normally negative in V V E
      • Tilt test
    • Management
      • Supine +/- feet up
      • Prevention
        • Pseudoephedrine
        • Metoprolol
        • Fludrocortisone
        • Disopyramide
        • Scopolamine
    • 2- Orthostatic Hypotension
      • What happen when we stand up?
      • HR, vasoconstriction
      • Absent or inadequate upright position response, Hypotension without increased HR
    • Diagnoses
      • BP and HR supine and standing up.
      • BP drop after 5-10 minutes up still by 10-15 mmHG
      • Positive tilt test without autonomic signs
    • Management
      • Elastic stockings
      • High salt diet
      • Corticosteroids
      • Slow upright position
    • Micturition Syncope
      • Rare form of orthostatic
      • Rapid bladder decompression associated with degreased total peripheral vascular resistance.
    • 3- Failure of systemic venous return
      • Increased intrathoracic pressure
      • Decreased venous tone (drugs; nitroglycerin)
      • Decreased volume (bleed…)
    • 4- Cerebrovascular occlusive disease
      • Mainly adult
    • Cardiac causes of Syncope
      • Structural heart disease
      • Arrhythmia
    • Why Cardiac ?
      • Syncope at rest
      • Provoked by exercise
      • Chest pain
      • Heart disease
      • FH of sudden death
    • What Cardiac
      • Obstructive lesions
      • Myocardial dysfunction
      • Arrhythmias
    • Obstructive lesions
      • AS, PS, HOCM, PHTX
      • Precipitated by exercise, no increase in cardiac output to accommodate increased demand.
      • Examination, CXR, ECG, Echo
    • Myocardial Dysfunction
      • Ischemia, infarction secondary to CHD, Kawasaki’s..
      • Myocarditis
    • Arrhythmia Arrhythmia Lack of output (Fast or slow heart) SVT, VT, SSS, CHB, Normal heart structure Long QT, WPW Abnormal Heart Structure Ebstein's, MS, MR, CCTGA Post op, TOF, TGA MVP VT Cmpthy SVT, VT, s brady
    • Long QT
      • Syncope, seizures, palpitation during exercise or with emotion
      • ECG
      • Ventricular arrhythmias (Tachy) with risk of sudden death
    • Long QT Defective ion channels Congenital Over 50 mutations in 4 sites Jarvell-lange-nielson Deafness AR Romano-ward no deafness AD Sporadic no FH no Deafness Acquired Drugs, illnesses, Autoimmune Neurological Nutritional Electrolytes
    • clinically
      • FH 60%
      • Deafness 5%
      • Presentation with Syncope 26%, seizure 10%, cardiac arrest 9%, presyncope palpitation 6%
      • Symptoms during exercise or emotion
      • Normally symptoms related to ventricular arrhythmias, mostly end of second decade of life.
      • Syncope in adrenergic arousal, exercise (swimming is a particular trigger)
      • Abrupt noises (Alarm, doorbell, phone..)
    • Tests
      • ECG with QTc >0.46 seconds
        • Frequently finding abnormal T wave
        • Bradycardia (20%)
      • Exercise test, maximum prolongation after 2 minutes of recovery, ventricular arrhythmia in 30% during exercise
      • Holter monitoring may show longer QTc
    • Diagnoses Criteria
      • Electrophysiological society
        • QTc >0.44 with no other causes (0.46 sec)
        • Positive family history plus unexplained syncope, seizure or cardiac arrest proceeded by trigger such as exercise, emotion
    • Treatment
      • Discuss with cardiologist
      • Avoid drugs associated with long QT
      • Avoid swimming, competitive sports
      • Beta blockers
      • Demand cardiac pacing (Pacemaker and defib)
      • Left cardiac sympathetic denervation
    • Prognoses
      • Untreated 75-80% mortality
      • Beta blockers reduce mortality to some extent
      • The adjusted annual mortality rate on treatment is 4.5% (10 year mortality of 50%)
    • Advise related to CHD
      • If one child has CHD, what are the chances of the second?
      • One parent has CHD, can offspring be affected? What are the chances?
      • See Handouts, statistical list of potential risks
    • Pathophysiology of congenital heart lesions
    • Pathophysiology of left to right shunt lesions ASD
    • Pathophysiology of left to right shunt lesions VSD
    • Pathophysiology of left to right shunt lesions PDA
    • Pathophysiology of left to right shunt lesions AVSD
    • Pathophysiology of Obstructive and valvular regurgitation lesions MR
    • Pathophysiology of Obstructive and valvular regurgitation lesions AR
    • Pathophysiology of Obstructive and valvular regurgitation lesions PR
    • Pathophysiology Cyanotic lesions TGA with good mixing RV 80% LV 90% 65% LA 90%
    • Pathophysiology TGA with poor mixing RV 45% LV 92% 100% 30% LA 92% 45% 45%
    • Pathophysiology TGA with poor mixing RV 45% LV 92% 100% 30% LA 92% 45% 45%
    • Tips
      • Read ECGs, easy to loose ECG skills.
      • Ask for help
      • As all specialties, it is only common sense.