Your SlideShare is downloading. ×
Pharmacology dysrhythmias 2
Upcoming SlideShare
Loading in...5
×

Thanks for flagging this SlideShare!

Oops! An error has occurred.

×
Saving this for later? Get the SlideShare app to save on your phone or tablet. Read anywhere, anytime – even offline.
Text the download link to your phone
Standard text messaging rates apply

Pharmacology dysrhythmias 2

990

Published on

Published in: Health & Medicine
0 Comments
0 Likes
Statistics
Notes
  • Be the first to comment

  • Be the first to like this

No Downloads
Views
Total Views
990
On Slideshare
0
From Embeds
0
Number of Embeds
0
Actions
Shares
0
Downloads
30
Comments
0
Likes
0
Embeds 0
No embeds

Report content
Flagged as inappropriate Flag as inappropriate
Flag as inappropriate

Select your reason for flagging this presentation as inappropriate.

Cancel
No notes for slide

Transcript

  • 1. Pharmacology Dysrhythmias Chapter 24
  • 2. Dysrhythmias• The electrical or conduction system controls the sequence of muscle contraction for optimal blood volume pumped from the heart each beat
  • 3. Dysrhythmias• Sometimes called arrhythmia• Occurs when there is a disturbance of the normal electrical conduction• Results in abnormal heart muscle contraction• Results in abnormal heart rates• The dangers is the frequency with which they occur• An inefficient pumping of adequate volume of blood• Certain type can produce additional dysrhythmias
  • 4. Dysrhythmias• Certain type can produce additional dysrhythmias – the heart stops pumping and still beat for a short time--fibrillations
  • 5. Dysrhythmias Classification• Origin within heart tissue – Electrical impulse sequence – Obstruction of conduction pathway – Combination – Based on heart beat rate
  • 6. Dysrhythmias• Causes:• Firing of abnormal pacemaker cells is when something causes out of regularity of electrical impulse (sequence)—irregular muscular contraction• Blockage of normal electrical pathways is a partial obstruction of normal conduction pathway causing an irregular pattern of muscle contractions—reentrant dysrhythmia• Combination of the both
  • 7. Sequential Causes• Above the Bundle of His—Supraventricular – Atrial flutter – Atrial fibrillation – Premature atrial contraction PAC’s – Sinus tachycardia – Sinus bradycardia – Paroxysmal supraventricular tachycardia
  • 8. Sequential Causes• Developing near or within the AV node are: – Junctional dysrhythmias• Developing below the Bundle of His are: – Ventricular dysrhythmia – Premature ventricular contractions—PVC’s – Ventricular tachycardia—VT – Ventricular fibrillation—VF
  • 9. Obstruction Causes• Described by location• Supraventricular• Ventricular• Left or right bundle branches• Atrio-ventricular blocks – Sub-classifications – 1st degree—partial block, delayed AV conduction – 2nd degree—partial block, with occasional blocked beats – 3rd degree—complete block • atria & ventricles function independent
  • 10. Dysrhythmias: Diagnosis• Physical examination• History• ECG pattern—underlying cause***
  • 11. Treatment of Dysrhythmias• The goal of therapy:• Restore normal sinus rhythm• Restore normal cardiac function• Prevent recurrence of life-threatening dysrhythmias
  • 12. Dysrhythmias: Drug Therapy• Anti-dysrhythmic are classified according to their effect on the electrical conduction system• Class Ia-IV – Sodium Channel Blockers (intermediate acting) – Sodium Channel Blockers (quick acting) – Sodium Channel Blockers (slow acting) – Beta Adrenergic Blockers – Potassium Channel Blockers – Calcium Channel Blockers – Vagal Stimulators – Conduction Slowers
  • 13. Dysrhythmias: Drug Therapy• Class• Ia• Ib• Ic• II• III• IV• Misc.
  • 14. Dysrhythmias: Drug Therapy• Class I act as myocardial depressant by inhibiting sodium ion movement
  • 15. Dysrhythmias: Drug Therapy• Class Ia prolong duration of the electrical stimulation on cells & refractory time between electrical impulses• Sodium Channel Blockers (intermediate acting)• Quinidine• Procainamide• Disopyramine
  • 16. Dysrhythmias: Drug Therapy• Class Ib shorten the duration of electrical stimulation & time between electrical impulses• Sodium Channel Blockers (quick acting)• Lidocaine• Mexiletine
  • 17. Dysrhythmias: Drug Therapy• Class Ic are the most potent myocardial depressant & slow conduction rate through atria and ventricles• Sodium Channel Blocker (slow acting)• Flecainide• Propafenone
  • 18. Dysrhythmias: Drug Therapy• Class II are BAB agents that depress the stimulation of beta cells of the SNS• Beta Blockers• Propranolol• Esmolol• metoprolol
  • 19. Dysrhythmias: Drug Therapy• Class III slow the rate of electrical conduction & prolong the time interval between contractions by blocking potassium channels• Potassium Channel Blockers• Amiodarone• Sotalol• Ibutilide• dfetilide
  • 20. Dysrhythmias: Drug Therapy• Class IV block calcium ion flow, prolonging duration of the electrical stimulation & slowing AV node conduction• Calcium Channel Blockers• Verampamil• Diltiazem
  • 21. Dysrhythmias: Drug Therapy• Misc Anti-dysrhythmia Agents• Digoxin—vagal stimulator• Adenosine—conduction slower
  • 22. Nursing Process: Dysrhythmias• Analysis & Diagnosis• Assess ECG• Telemetry in place as adjunct• 24 hour ECG Holter monitor• EPS - electrophysiologic studies• Exercise electrocardiography• Lab & DIAGNOSTIC TESTS: K+, Ca+, ABG, clotting: AST, ALT, CK-MB, creatinine troponin, D-diameter, LDH, X-ray, cardiography, catherization• Examine urinalysis & perform hourly I & O’s• Report < 30-50mL urine output• Monitor renal function—BUN and creatininte• Medical history—meds prescribed• 6 Cardinal Signs of CVD (dyspnea, chest pain, fatigue, edema, syncope, palpitations• Mental status: LOC, clarity of thought—cerebral perfusion• VS: BP twice daily, record BP in both arm 5-10 mm Hg or more reported, HR, RR, temperature, O2 sat, BrS• Auscultation & Percussion for heart size and sounds
  • 23. Nursing Process: Dysrhythmias• Decreased Cardiac Output• Activity Intolerance• Risk for Decreased Cardiac Tissue Perfusion• Fatigue• Death Anxiety
  • 24. Nursing Process: Dysrhythmias• Planning:• Order medication & place in the MAR• History of 6 Cardinal Signs of CVD• Basic Mental Status once per shift• VS, Auscultation & Percussion schedule consistent with client’s status• Lab test order stat & subsequent labs; monitor result• Emergency treatment: “codes”—cardioversion and defibrillation
  • 25. Nursing Process: Dysrhythmias• Implementation:• Monitor ECG telemetry• Perform physical assessment (Q4-8 hr)• Assist with ADL’s, note the degree of impairment or dyspnea w/or w/o exertion• O2 as ordered or PRN• Administer prescribed meds & ACLS protocol to best alleviate symptom for maximum comfort• Encourage physical activity• Measure to reduce anxiety
  • 26. Nursing Process: Dysrhythmias• Education & Health Promotion:• Identify modifiable factors• Design approach to modify factors within client’s control• Teach, Discuss & Practice coping mechanism to handle anxiety• Teach to take pulse rate, blood pressure, signs & symptoms
  • 27. Nursing Process: Dysrhythmias• Fostering Health Maintenance:• Discuss med information & how it will benefit• Discuss meds should be taken as prescribed• Provide client & significant other w/important information from drug monograph• Teach drug adverse effects and how to respond• Seek cooperation & understanding of:• Name of med• Dosage• Route• Times• Common and Serious adverse effects
  • 28. Nursing Process: Dysrhythmias• Written Record:• Enlist client’s aid in developing & maintaining a written record• Monitoring parameter to include like HR, BP, degree of dyspnea & precipitating causes, chest pain, edema• Ensure client know how to use form• Instruct client to bring completed form to follow up visits

×