Drug addiction neurobiology
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Drug addiction neurobiology

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  • CRF imp n depenence n withdrawl.description f extended amygdala later on.
  • CRF imp n depenence n withdrawl.description f extended amygdala later on. sites---??, neurotransmitters--??
  • Alcohol- increase gaba, reduce glutamate, on VTA, the opiod receiptor s meu R., in NAc, the opiod receiptor s kappa receiptor.
  • Nxt will b CRF
  • Voltage operated ca channels, N methyl D asperate, ca entry in cell reduced. As ca s imp for neurotransmitter release.so there will be less release
  • glucose utilization (yellow represents less utilization and blue shows the least). The right scan is taken from a cocaine abuser on cocaine. It shows that the brain cannot use glucose nearly as effectively—show the loss of red compared to the left scan. There are many areas of the brain that have reduced metabolic activity. The continued reduction in the neurons' ability to use glucose (energy) results in disruption of many brain functions.
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Drug addiction neurobiology Drug addiction neurobiology Presentation Transcript