Leucoderma & its Homoeopathic Management


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  • Cold weather can aggravate the condition...it is not beneficial for a leucodermic patient.
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  • One another question, right now i am in India but i wants go out of India if i got job there. So, If i go for Canada then canada's atmosphere will work to cure Leucoderma?

    If people stay in cool atmosphere, is it beneficial for them to cure that?
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  • Any allergic disease does not cure permanently. We can just increase the immunity which will help the patient not to suffer with great intensity. As soon as any exciting factor comes it will again disturb the vital force of the individual.
    These disease mostly occurs only when there is genetic dyscrasia.
    From past experiences I have not seen any effect of Crab Soup.
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  • Is Crab Soup Beneficial in Lucoderma? If Lucoderma genetically present then is it possible to cure permanently?
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Leucoderma & its Homoeopathic Management

  2. 2.  Leucoderma is a general term applied to decreased melanin pigmentation of skin.It is a cutaneous condition,which includes all depigmented (white) lesions of skin.
  3. 3. ETIOLOGY OF LEUCODERMA  IDIOPATHIC  CHEMICALS : Vitiligo : Substituted phenols (used in foot wear) adhesives (used in stick-on bindis)  INFLAMMATORY SKIN DISEASES : Lupus vulgaris discoid lupus erythematosus ( Ref: Synopsis of Dermatology by Neena Khanna)
  4. 4. Vitiligo is a continual and long term skin problem that produces white depigmentation patches that develop and enlarge only in certain sections of the skin. The term Vitiligo is derived from the latin word Vitilus - meaning calf and was first used by roman physician Celsus of 1st century AD.The characteristics white patches of disease resembled the white patches of a spotted calf in India.
  5. 5. EPIDEMIOLOGY 1.Incidence-occurs in about 1% of population. 2.Gender-no gender predilection 3.Age – affects all ages,peak incidence between 10-30years.
  6. 6. ETIOLOGY OF VITILIGO  Autoimmune: Autoimmune disorders like Thyroid disorders (Hashimoto thyroiditis and Graves disease), Addison disease, diabetes mellitus, alopecia areata, pernicious anemia, and psoriasis are all associated with Vitiligo.  Genetic: About 20% of affected patients have a positive family history of Vitiligo.  Neural Theory: segmental Vitiligo is present along a dermatome in distribution of nerves.It has been hypothesized that a toxin,which destroys melanocyte,is released at the nerve endings.
  7. 7. HISTORY 1. There is a positive family history in 20% of patients. 2. Both sexes are affected equally. 3. Half of cases begin before age of 20 years. 4. Patient may state that the first onset occurs after emotional stress,trauma,illness. 5. It progresses slowly over the years in a highly variable course. 6. Some patients have very stable disease , in others, it progresses at a alarming rate.
  9. 9. CLINICAL FEATURES  MORPHOLOGY: characterised by depigmented macules, which are chalky or milky white. sometimes,pigment loss is partial and occasionally, three shades (trichrome).
  10. 10.  Hairs in the lesions may remain pigmented, though in the older lesions the hairs may lose their pigment (leucotrichia).
  11. 11.  Macules have a scalloped outline and form geographical patterns on fusion with neighbouring lesions.
  12. 12. SITES  De-pigmentation appears on the sunexposed areas. Common sites affected include: • Bony surfaces -back of hands and fingers, elbows and knees.
  13. 13.  Around body orifices - around the eyes, mouth and nose.  Body folds - armpits and groins.  Others areas -
  14. 14. PATTERNS VITILIGO VULGARIS     Commonest type. Occurs after second decade. May be slowly or rapidly progressive. Family history is frequently present. SEGMENTAL VITILIGO     Occurs in children. Most frequently(50%) seen in distribution of trigeminal nerve. Has a stable course. Margins are feathery.
  15. 15.  GENERALIZED VITILIGO variants of generalized vitiligo are:  Acrofacial Vitiligo: This type of vitiligo is considered only when the appearance is mostly on the fingers or toes.  Mucosal Vitiligo: The appearance of the depigmentation generally around the mucous membranes and lips.  Focal Vitiligo: one or a few scattered white patches in a small certain areas. It is often noticeable in young children.
  16. 16. How will you confirm whether a white spot is leucoderma? • If white spot has an outline darkness than the skin. • If it slowly and gradually increases in size day by day. • If it is irregular in appearance. • If appears milky white in color under an ultraviolet light. • If appears red in color in mucus membrane.
  17. 17. How is Leucoderma Diagnosed?  Age of onset(usually not present at birth).  Depigmented macules with scalloped borders.  Leucotrichia  Predilection for sites of trauma.  In addition,medical and family history.  Biopsy of the affected area can confirm the diagnosis of Vitiligo.
  18. 18.  Blood tests done:  To check the thyroid functions  To look for the presence of anti-nuclear antibodies (that would point towards auto-immune cause)  To rule out pernicious anemia  Examination of the spots under Wood's lamp can be used to diagnose and evaluate Vitiligo especially when the changes are not appreciated with the naked eye.
  19. 19. DIFFERENTIAL DIAGNOSIS:  Nevus achromicus  Pityriasis alba  leprosy  Tinea versicolor  Albinism
  20. 20.  Nevus achromicus  Onset-present at birth  Distribution- segmental/focal  Morphology-feathered margins,uniform pigment dilution  Hair- no leucotrichia
  21. 21.  Pityriasis alba  Morphology- scaly hypopigmented macule  Sensation-Normal  Nerves- not thickened • Leprosy  Hypopigmented/ertythematous macules,nodule,plague may be present.  Sensation- less  Hair- decrease in no
  22. 22.  Tinea versicolor  Located on upper trunk,less frequently on forearms/face.  Morphology-small perifollicular,scaly macules which may become confluent.  Sensation- normal. • Albinism  It is an autosomal recessive disorder  At birth,there is complete absence of pigment in skin, hair & eyes.
  23. 23. What is the Homoeopathic approach ?  As a rule, Homoeopathy never looks at Leucoderma as a local disease. Leucoderma has been considered as a local expression of a system disturbance hence a constitutional remedy has to be evolved, which calls for individual case study in every case of leucoderma. That is why there is no single specific remedy for all the cases of leucoderma. The exact treatment is determined only on in-depth evaluation of individual case.
  24. 24.  Like all other chronic diseases, two factors are necessary to cause leucoderma—  (i) Inherited and / or acquired miasmatic dyscrasia  (ii) Environmental factors in the form of physical trauma, chemical irritants, emotional upset etc.  No true aetio-logical factors have yet been demonstrated nosologically but a homoeopath knows that all true chronic diseases are nothing but the products of three basic miasmatic states, — Psora, Sycosis and Syphilis. The aetiologieal factors mentioned in the text books of medicine for skin diseases are but precipitating factors. Unless the basic miasmatic state is there, these environmental factors can never cause leucoderma.
  25. 25. Miasmatic Diagnosis
  26. 26.  Complete absence or diminution of melanocytes from the very birth is due to Syphilitic miasmatic state and requires antisyphilitic medicines for cure.  Cases where the function of the melanocytes is defective resulting in relative melanin deficiency is due to Sycotic miasmatic state as it represents a state of in coordination.  Secondary depigmentation during the course of an illness may be due to Psora or Sycosis or Syphilis or a combination of two or three of them.  This can easily be elicited from the past history, personal history -and family history of the patient.
  27. 27.  Homoeopathically, Leucoderma is a one sided disease having its-chief or only manifestation on the skin to start with.  In aphorism no.185, Dr. Hahnemann discussed about one sided disease with local manifestations.
  28. 28. ONE-SIDED DISEASES (Diseases with too few symptoms and chiefly of chronic nature) Principal symptoms Internal Complaint Physical Complaint Mental Complaint External Complaint Local Diseases or Local Maladies
  29. 29. Local Disease  A kind of one sided disease in which the changes and ailments appear locally on the external parts of the body. External cause Arising from external lesions Internal cause Arising from the internal malady
  30. 30.  Naturally, it is very difficult to cure, because we do not get the exact-sensations, modalities or concomitants in such cases. But if we-believe in miasmatic concept of diseases and treat these patients with suitable antimiasmatic medicines, supplemented by proper hygienic measures and removal of the possible obstacles in the way of cure, then, in most cases, we may be able to cure these patients.  But it must be remembered that melanisation of the patches from where melanocytes have completely disappeared is a most difficult task to perform and requires a prolonged course of treatment and patience both on the part of the patient and the physician.
  31. 31. Plan of Treatment
  32. 32.  Whatever may be the nature and type of Leukoderma, we must first of all, try to ascertain the miasmatic background of the patient. Once this is done the major problem is over.  The next step is to select a medicine which covers the constitutional totality of the patient including the miasmatic background.
  33. 33.  At the same time we must take care of the environmental or precipitating factors. Unless the exciting factors are properly dealt with, no cure, in the truest sense of the term, is possible in spite of proper antimiasmatic treatment. The reason is obvious. The presence of these factors act as obstacle in the formation of melanocytes.  Vaccinosis in the past and abuse of drugs must be antidoted as and when necessary during the course of antimiasmatic treatment.
  34. 34.  Frequently, a change in the plan of treatment is necessary to effect a complete cure (e.g. antisyphilitic treatment followed by anti-psoric or anti-sycotic as the case may be followed again by anti-syphilitic and so on) In this way a prolonged course of treatment (may even be for years together) is absolutely necessary.  Any attempt to remove only the patches of leucoderma by Homoeopathic medicine is bound to be futile as far as this is the reason why we fail in Homoeopathy to cure such cases.
  36. 36.  Formation of complete picture by noting-  The exact character of local affection.  All the changes, sufferings, symptoms observable in patient’s health.  Symptoms which may have been previously noted when no medicine had been used.
  37. 37.  Selection of Homoeopathic medicine whose peculiar pathogenetic effect corresponds to the totality of symptoms of the case.  External application is prohibited even though it is specific Homoeopathically.  External treatment is always injurious.
  38. 38. Potency and Repetition of the selected drug
  39. 39.  LM potencies yield best results in such cases as there is less chance of aggravation and they may be repeated safely for a fairly long period of time.
  40. 40. A word of Caution
  41. 41.  We must not be afraid of a few newly depigmented areas during the course of treatment because these may occur due to the action of well indicated medicine and should be considered as a favourable sign instead of considering it a bad sign provided the old spots must, at the same time, show some signs of improvement as mentioned above or the patient as a whole feels better.
  42. 42. Self Care Measures with Diet and Regimen
  43. 43.  It is also no less important to make care of diet and other hygienic precautions.  Lemon, sour and citric acid containing food stuffs are to be avoided as they may act as hindrance in the way of cure.  Some studies show that avoidance of meat may expedite cure though the reason is not yet clear.  Use of cosmetics (especially the use of lipstick, vermilion etc.) is to be avoided or restricted as a routine.
  44. 44.  Assurance: The patient must be assured that the disease is not at all incurable and not infectious or contagious. Otherwise, persistent mental anxieties and worries due to wrong conception of the disease will doubtlessly act as an impediment in the way of cure Hence the patient should be assured that the disease is not a sin and there is nothing to be ashamed of.  From Homoeopathic view point these cases are never to be treated with local medicines because that will lead to suppression and, consequently, destruction of some of the vital organs.
  46. 46.  Expose the affected areas of the body to the early morning sun for 20-30 minutes daily.  Use a mild soap while bathing.  Avoid using any cosmetic applications on skin.
  47. 47.  Make sure that your diet contains enough iron, cereals, beans and green leafy vegetables which are some of the best sources of iron.  Avoid consuming sour foods, citrus fruits, fish,red meat, crabs and prawns.  Use of synthetic clothes should be restricted.
  48. 48.  Avoid steroids.  Avoid stress and learn to relax.  Keep a positive attitude.
  49. 49.  Thus, it is possible to be leucoderma free with Homeopathic treatment, hence one must consider Homeopathy for healthier living.
  50. 50. For LEUCODERMA
  51. 51. ACCORDING TO KENT’S REPERTORY: SKIN-DISCOLORATION- -WHITE:Apis,Ars,Kali.c :fl-ac -SPOTS:Ars,sil. :Calc,Carb-v,Lac-c,Sumb :Alum,Aur,Berb,Calc,Merc,Nat.c,Phos ,Sep,Sulp. :Am.c,Carb.an,Coca
  52. 52. ACCORDING TO BOERICKE REPERTORY: Leucoderma- -Ars.s.fl -Nat.m,Nit.ac,Sumb
  53. 53. Recent CCRH Research For Leucoderma In Clinical Research Publication in Indian Journal of Homoeopathic Research in Homoeopathy CCRH. -Dr. Ravi Kumar -A Case of Vitiligo treated with “ PHOSPHORUS” (Indian Journal of Research in Homoeopathy 2008;page-49-57) -Dr. Jha , Dr. L.Debata-A Case of Vitiligo treated by “SULPHUR” (Indian Journal of Research in Homoeopathy 2009;page-34-40) …………((website-ccrhindia.org/list%2of%20publication.as))
  54. 54. Cont.........  Clinical Research Publication Book of CCRH-Clinical Research series-1,2,3 -Dr.R.V.R Prasad,Ch.Ravendar,Bharatha Lakshmi k singh,Dr.A.K Vichitra,Praveen Oberai,Renu Mittal -Clinical Evaluation of Predefined Homoeopathic Medicines in Vitiligo. (Clinical Research studies series-ii,2009,page-85-92.) .......(website-ccrhindia.org)
  55. 55. Specific Remedies (Ars.sul.fl , Ars.iod) Homoeopathic Remedies Constitutional Remedies (Kali.c,Cal.c,Apis.m, Nat.m,Nit.ac,Gel.) InterCurrent Remedies (Thyr.,Tub.)
  56. 56. Arsenicum Sulfuratum Flavum(Ars.s.fl)  Good remedy for Leucoderma  Squamous Syphilides.  Skin chafed about genitals.
  57. 57. Arsenicum Iodatum  Skin-Dry,Scaly,Itching.  Marked exfoliation of skin in large scales,leaving a raw      exuding surface beneath. Ichthyosis Debilitating night sweats. Emaciation. Acne hard,shotty,indurated base with pustule at apex. Acrid excoriating discharge.
  58. 58. cont……  Modalities- < weather, dry cold, windy foggy, cold wind, cold bath, wet weather, exertion, lying on painful side, warmth, warm room, warm wraps, walking fast, exercise, during menses, when Hungry, Apple,Tobacco,Smoke.
  59. 59. cont…… > from eating, open air, Rest, uncovering.
  60. 60. Apis Mellifica Swelling or Puffing up of various parts.  Oedema  Red rosy hue, Burning sensation  Stinging pain  Soreness  Intolerance of heat-Warm blooded(worse in warm and closed room  Intolerance of slightest touch(agg.from touch)
  61. 61. cont……..  Awkwardness (let things fall from hands)  Right sided troubles  Modalities- < Heat in any form, touch, pressure, late in afternoon, in closed and heated room rt.side > in open air, uncovering, cold bathing.
  62. 62. Kali-carb.  Chilly(worse by cold)  Wants company(aversion to being alone)  Rt.sided  Inclined to obesity  Lying on painfull side aggravates.  Broken down constitution(pale,anaemic,poor digestion,acidity,dyspepsia,neuralgia,dropsy)
  63. 63. cont……….     Sharp , cutting pain. Stinging pain in muscles. Burning as from a mustard plaster Modalities < after coition, in cold weather, from soup and coffee, in morning(about 3’o clock), lying on lt. side and painful side > warm weather, though moist, during day, while moving about.
  64. 64. Carbo-Veg.  Disintegration.  Imperfect oxidation(low vitality)- feeble circulation.  Blue,cold ecchymosed.  Marbled with venous over distension.  Itching <on evening,when warm in bed.  Moist skin.  Perspiration- Hot.  Offensive discharge.
  65. 65. cont……….  Modalities- < evening , night, open air, from fat food,butter,coffee,milk, warm, damp weather, > eructations, from fanning, from cold.
  66. 66. Natrum-Mur.           Face-oily , shiny , as if greased. Liable to take cold. Craving for salt. Worse from over exertion in sun Weeping tendency. Consolation aggravation. Cannot bear heat of sun. Ailment with silent grief. Hypochondriac anxiety. Mental exertion aggravates.
  67. 67. cont………  Fear of Thunderstorm.  Indifference  Ailments from mortification.  Modalities- < noise , music, warm room, lying down, abt.10 a.m at seashore,
  68. 68. cont………. mental exertion, consolation, heat talking, > open air, cold bathing, going without regular meal, lying on rt. side, pressure against back, tight clothing.
  69. 69. Tuberculinum Intense Itching.  Worse at night.  Tuberculosis in their Family.  Light complexion,blue eyes,tall,slim, narrow chested,active & precocious mentally,weak physically.  Susceptible to cold.  Emaciation on while eating well.
  70. 70. cont…………..  Modalities- < Motion, music, before a storm, standing, dampness, droughts, early morning, after sleep. > open air.
  71. 71. Thyroidinum         Stupor,alternating with restless melancholy,irritable. Worse least opposition,goesinto a range of trifles. Psoriasis associated with adiposity. Skin-dry,impoverished. Cold Hand and Feet. Browny swelling. Itching without eruption < at night. Excessive obesity. Marked sensitiveness to cold Hypothyroidism after acute dis,ie.weakness.  Easy fatigue  Weak pulse
  72. 72. cont………………  Tendency to fainting,palpitation,cold hand and feets,low B.P,chilliness & sensitive to cold.  Modalities< cold, cold weather, sexual excesses, during menses, during pregnancy, during labour, during puerperal period, during menopause. > lying on abdomen.
  73. 73. Bibliography Synopsis of Dermatology       -By KHANNA NEENA Skin Disease -By P.HABIF THOMAS Organon of Medicine(5th and 6th Edition) -By DUDGEON R.E Lectures on Homoeopathic Materia Medica -By KENT J.T Pocket manual of Materia Medica and Repertory -By BOERICKE WILLIAM Systemic Materia Medica of Homoeopathic Remedies -By MATHUR K.N Website- ccrhindia.org/list%2of%20publication.as
  74. 74. Thankkkk uuuuuuuu …….