Piroplasma dr reda e khalafalla
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Piroplasma dr reda e khalafalla

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Piroplasma dr reda e khalafalla Piroplasma dr reda e khalafalla Presentation Transcript

  • Characteristics: Intracellular blood parasites Absence of conoid, oocyst, spores, pseudocyst, flagella, no hemozoin pigment Gametocytes are found in erythrocytes or lymphocytes Heteroxenous i.e merogony by binarry fission and budding occure in vertebrate host (asexual) while gametogony and sprogony (sexual) in invertebrate host (hard tick) Locomotion by body flexion or gliding Class II: Piroplasm Class: Piroplasmorida Family: TheleriidaeFamily: Babesiidae Family: Anaplasmiidae genus: Theleriagenus: Babesia genus: Anaplasmia
  • Genus: Babesia Stage to stage transmission Transovarian transmission
  • Babesia spp. Life cycle • Schizogony/ merogony occurs in RBCs • B. Equi (T. Equi or Nuttalia equi) merogony occurs in lymphocytes • In RBCs the parasite is formed by union of two merozoites except B. Equi are four merozoites (cross-shaped)
  • Sporogony n-times Gamogony Merogony Host VECTOR TICK heteroxenous
  • Host (IH) species VECTOR Cattle B. divergens Ixodes ricinus B. major Haemaphysalis punctata B. bigemina Rhipicephalus microplus B. bovis Dog B canis Dermacentor reticulatus B. gibsoni Haemaphysalis bispinosa u.a. Horse B. caballi Dermacentor spp. T. (B.) equi Dermacentor spp. (Nuttalia equi) Rhipicephalus spp. Hyalomma spp. Human B. microti Class II: Piroplasm
  • Pathogenicity of Babesia spp. • Babesiosis or Red water or Taxas fever or Tick fever----highly pathogenic---death • Body temprature 41°C, rouphy coat, restlessness • Acute formHeamoglobinuria (destruction of RBCs) Jaundice, aneamiaspleenomegaly liver and kidney damage • Chronic form emaciation, no clinical signs • In human signs are similar to malaria • Diagnosis: case history, blood film, serological diagnosis • Treatment: Imizol, Berenil, Acaprine and supportive treatment • Tick control program
  • Theileria annulata, T. parva • Absence of transovarian transmission only stage to stage transmission (interstadial transmission) • Schizogony/merogony occurs in the lymphocytes • Infection from tick by sporozoites, coma-shaped, enter the lymphocyts • form Koch‘s blue bodies (schizont)..... Rupture of lymphocytes another infection to lymphocytes ..... • Forming the macro and microgametocytes-- taken by tick during blood meal • --- ray bodies- fusion-- motile kinete (zygote) penetrate the intestinal wall of tick- oocyst , Large No. Of sporozoites ...migrate to salivary gland...infect another host
  • Theileria
  • Pathogenicity of Theileria spp. Theileria annulata Egyptian fever or Mediterranian cost fever affect cattles, sheep, Ox, water buffaloes Acute form fever 41°C, decrease in milk production, swelling of superficial lymph nodes and eye lids, bloody diarrhia, anaemia, jaundice Chronic form irregular fever, inappetance, emaciation, anaemia, enllargement of lymph nodes T. parva cause African coast fever or Bovine theileriosis or East cost fever affect water buffaloes and cattles Signs same to T.annulata + internal haemorahgic foci, ulcers in the abomasum T. mutansBovine gonderiosis T. camelensiscamels T. ovis  sheep and goats
  • Babesia major Babesia canisBabesia divergens Theileria sp. Intraerythrocytic Stages of Piroplasms
  • Babesia-Stages in Tick gut
  • Babesiosis: internal Bleeding
  • Babesiosis: Bloody urine
  • Piroplasmosis in Horses
  • Control measures of Babesiosis Treatment (Imidocarb, Berinel inj., symptomatic treatment) Chemoprophylaxis (Imidocarb) Tick control and prophylaxis