Varicose Veins


Published on

Published in: Health & Medicine
  • Be the first to comment

No Downloads
Total Views
On Slideshare
From Embeds
Number of Embeds
Embeds 0
No embeds

No notes for slide

Varicose Veins

  1. 1. Varicose veins and treatment• Jeannouel van Leeuwen , surgeon• Chirurgen Maatschap Emma Care• Courtesy of Servier• 25 january 2012
  2. 2. What we‟ll cover• Some Definitions• Anatomy• What are you looking for?• Examination techniques• Treatment options
  3. 3. Incidence• annual incidence of varicose veins is about 2%• life-time prevalence of varicose veins approaches 40%• Varicosities are more common in women (about 2-3 times as prevalent in women than in men)• 10-20% actually are symptomatic enough to complain about their lower leg varicose veins and seek treatment.
  4. 4. What is a varicose vein?• Long, tortuous and dilated vein of the superficial varicose system• Commonly legs but where else? • Abdominal Wall • Anus • Vulva • Oesophagus • Scrotum
  5. 5. Why do they happen?• increased pressure in the superficial venous system• normally blood flows from superficial system to deep• if the valves protecting the superficial veins become incompetent there is higher pressure in the superficial veins and they become varicose
  6. 6. Normal venous flow in the LegNormal Flow • Superficial veins drain into the deep veins •From the foot up to the heartSuperficial vein disease always starts with abnormalvalves and interruption to normal flow called venousreflux
  7. 7. Abnormal flow = Venous RefluxDamaged Valves 1. Blood flows to the skin 2. Blood is pushed distally and proximally 3. Close loop recirculation 4. Blood is retained in the leg • Increased volume of blood (heaviness Fatigue) • Increased venous pressure • Veins Dilate (varicose veins)
  8. 8. Taking the history Presenting Complaint: Varicosities, abdominal/groin lump – saphena varix Symptoms  Localized discomfort in the leg, Pain, Swelling, Venous claudication, Itching “Risk” factors  Female, age, ethnicity, occupation, pregnancy, obesity, sm oking  ASK about history of abdominal complaints/cancer, DVT, previous & other venous complaints
  9. 9. So the examination• Inspection• Auscultation• Palpation • cough test • tap test• Tourniquet Tests • Trendelenberg • Tourniquet test • Perthes• Doppler • Sapheno-femoral junction • Sapheno-popliteal junction
  10. 10. Diagnosis of venous disease• Physical exam • Appearance • Trendelenburg test • Palpation • Hand Doppler• Duplex Examination • R/O DVT • Size of veins • Map out superficial veins • Locate the site of reflux • Reflux 0.5 sec in GSV and 1 sec in deep system • Find refluxing perforators
  11. 11. Clinical picture - symptoms• Cosmetic disfigurement• Pain and discomfort• Night cramps• Mild swelling at night• Pigmentation• Itching• Ulceration
  12. 12. Anatomy• Superficial System arises from foot and ends at Sapheno- femoral junction (spiderhead)• Long saphenous vein- medial leg up to SFJ• Short saphenous vein- lateral malleolus , up calf to meet popliteal vein behind knee• Sapheno- femoral junction- 4 cm lateral and 4cm below the pubic tubercle• Communication veins: connecting deep and superficial system through piercing deep fascia, with valves to direct blood from superficial to deep viens.• Perforator veins: there are 3 perforators on the medial side and 1 on the lateral side of the leg
  13. 13. Inspection- other features1. Spider Veins- blueish vessels that distend above the skin surface2. Thrombophlebitis- superficial red painfull lump3. Brown pigmentation- haemosiderin deposition4. Venous Eczema5. Venous Ulcers- over medial ankle6. Lipodermatosclerosis-progressive sclerosis of cutaneous fat- ankle becomes thin and hard- area above becomes oedematous7. Scars from previous surgery
  14. 14.  Atrophy blanche  Ulceration: active and healed Inspection  Leaves a white patch Venous ulcers/eczema  Pitting oedema Spider veins
  15. 15. Inspection Lipodermatosclerosis  Literally "scarring of the skin and fat“  A slow process that occurs over a number of years and has 2 phases:1. Acute  Venous pooling →chronic venous hypertension  RBC forced into surrounding tissue  Haemoglobin broken down into brown haemosiderin2. Chronic  Chronic haemosiderin formation leads to fibrin deposition  Skin becomes thickened and shiny  Skin around ankle constricts and the inverted champagne-bottle shape is seen
  16. 16. Stages of chronic venousinsufficiency(Expert meeting in Moscow, 2000.)• 0 - no symptoms;• 1 - heavy feet syndrome;• 2 - intermittent edema;• 3 - persistent edema, hyper- or hypopigmentation, lipodermatosc lerosis, eczema;• 4 - venous ulcer.
  17. 17. CausesPrimary• Theories of Aetiology: • Weak wall theory • Congenital valvular incompetence• Aggravating factors: • Female sex • High parity • Occupation requiring prolonged standing • Marked obesity • Constricting clothes • Estrogen intake • Deep venous thrombosis
  18. 18. SecondaryAnything that raises intra-abdominal pressure orraises pressure in superficial/deep venous systemso…: •Pregnancy •Abdominal/pelvic mass •Ascites •obesity •constipation •thrombosis of leg veins (DVT) •AV fistula •Vena cava thrombose •Large liver cysts
  19. 19. Auscultation• Auscultate over any varicosities for bruits• due to A-V malformation
  20. 20. Palpation• Palpate the veins to confirm they are infact veins- will refill if if gently pressed and released• Next- find the sapheno-femoral junction (SFJ) • Find Pubic Tubercle just lateral to pubic symphisis • 4 cm lateral then 4cm below • Palpate for a sapheno varix- localised distension of the long saphenous vein in the groin• Cough Test- Fingers over SFJ, ask patient to cough can you feel a thrill, if yes suggest incompetence• Tap Test- tap over the SFJ and feel further down long saphenous vein for any transmitted sounds, if yes suggest incompetence
  21. 21. Trendelenberg/Tourniquet testsAim- to localise the valve/s that are incompetentTrendelenberg• Lie patient down and raise leg attempting to drain varicosities of blood.• Using either a tourniquet or fingers put pressure over SFJ to occlude it• Ask patient to standIf varicosities DO NOT refill indicates SFJ incompetenceIf DO refill the leaky valve is lower down„I will now try and locate the incompetent perforator using the tourniquet test‟
  22. 22. Tourniquet test continued• Same as before- lie down, raise and drain leg• Place tourniquet approximately over area of each perforator( mid thigh, sapheno popliteal, calf perforators)• If varicosities DO NOT refill that perforator is incompetent• If varicosities DO refill continue down leg
  23. 23. To complete my examination Iwould like to…• Perform a full Abdominal Examination• Scrotal examination ( on males!)• Arterial ExaminationInvestigations• Duplex Ultrasonography- maps valve incompetence• Phlebography not done anymore
  24. 24. Spider VeinsThe proper term is Telangiectasia •These are non raised dilated veins located in the Dermis (deep layer of the skin) •Single layer endothelium, minimal muscle •Can be Red or Blue in color depending on the origin •Do not cause major medical complications •Appears earlier than varicose veins (4% of teenagers , and 13 % in 18 to 20 year olds •More common in females •Reticular Veins are lager feeding veins
  25. 25. Spider VeinsEtiology: Multifactorial • Venous Hypertension associated with varicose veins • Congenital: vascular nevi, neonatal hemangiomatosis, others.. • Collage Vascular Disease: lupus, • Hormonal factors: pregnancy, estrogen therapy, topical steroids • Trauma: contusion, incisions • Infections
  26. 26. Venous Stasis Ulcers• Differential Diagnosis 1. Venous ulcerations 50% on non healing ulcers 2. Arterial ulcers in about 10% 3. Malignancy : basal and squamous cell, lymphoma 4. Infections: HIV, fungal 5. Collagen vascular disorders: Lupus ec. 6. Lymphatic obstruction• Affects over 1 million people in the US• 100,000 are disabled from this• More common in elderly population
  27. 27. Ulcus cruris venosum
  28. 28. Venous Stasis Ulcers• Etiology 1. Venous Hypertension • Venous reflux • DVT • Varicose veins 2. Edema 3. Biological factors • Leakage of proteins impedes diffusion O2 • Aggregation of white cells • Block capillary flow • Release on inflammatory proteins
  29. 29. Management  Surgical Conservative/Medical • Ankle-to-groin saphenous vein  Graded compression stripping (with stab avulsion) bandaging, Compression • Segmental saphenous vein stripping hosiery (with stab avulsion) • Saphenous vein ligation:  Paste Gauze (Unna) Boots high, low, or both  Diuretics? Zinc? • Saphenous vein ligation and Phlebotrophic/Hemorheologi sclerotherapy c agents? Aspirin/NSAIDs etc • Saphenous vein ligation (with stab avulsion) • Stab avulsion of varices without saphenous vein stripping (phlebectomy) • Endoluminal occlusion of the saphenous vein by radiofrequency (RF) or laser energy
  30. 30. Surgical ligation and Stripping • Standard treatment for a century • General anesthesia • Pain • Long recovery • Some complications • Good cosmetic results
  31. 31. Surgical treatment• Crossectomy or/and vein stripping till below knee better than compressive therapy alone• Other techniques : Endovas.burning or foam injection
  32. 32. Vein Ablation• Laser Ablation (EVLA ) • Uses light to heat the vein• Radio Frequency (VNUS Procedure) • Uses radio frequency to heat the vein • Office based procedure • Done under local anesthesia • One needle puncture at the level of the knee • Takes about 1 hour • Patient resumes normal activity same day
  33. 33. EVLA Results Images from
  34. 34. Sclerotherapy• Cumulate vein with needle• Inject Sclerosing Solution • Ethoxysclerol • Hyper tonic Saline • Foam (Mix STS with air and make bubbles)• Intravenous injection causes intima inflammation and thrombus formation
  35. 35. Sclerotherapy Use• Neovascularization• Perforators• Clean up after Phlebectomies• Spider veins• Reticular veins• GSV: can closure the, but has high recurrence rate
  36. 36. Sclerotherapy results
  37. 37. UNNA bootresult • Weekly change with UNNA boot bandage gives nice result
  38. 38. • Compressive bandages first choice with simple small vein ulcer
  39. 39. • Skin grafting can be put on a non infected granulating skin defect of a venous ulcer
  40. 40. Treatment complications• Major complications following VV surgery are relatively rare• Up to 20% morbidity • Infection • Hematoma • Pain • Nerve damage • Saphenous nerve (LSV surgery) • Sural, peroneal nerve (SSV surgery) • Lymphatic leak - Venous thrombosis - Vascular injury • Recurrence
  41. 41. Oral medication• Effect on edema , hematocrit , augmentation capillary permeability , inflammation , less fibrinolysis , leukocyte function en erythrocytes• No evidence for monotherapy only in addition effect on ulcer healing• Daflon , Trental , Aspirine
  42. 42. Рhlebotropic drugs• Daflon• Venal• Venoruton• Doxium
  43. 43. Rheologichemocorrectors• acetylcalicylic acid,• dipiridamol• pentoxyphylline• low-molecular dextranes
  44. 44. Thank you for your attention www.curacaoveininstitute.comChirurgen Maatschap Curacao