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Infection   oms (1)
 

Infection oms (1)

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    Infection   oms (1) Infection oms (1) Presentation Transcript

    •  Infection  Trait  Arising  Mutation  Diagnosis  Treatment Conception Anatomy Route Influence factors Principles Principles
    • Infectious agent(bacteria) Host Inflammatory reaction protective and defensive Beneficial Elimination the infectious pathogen Repair tissue injury Harmful Hypersensitivity Autoimmune disease
    • Oral and nasal cavity  Tooth  Space  Blood and Lymphoid system  Bacteria 
    • Bacteria---Exist Oral and nasal cavity  Maxillary sinus  Temperature + Moisture (Beneficial) Reproduction + Developing
    • Tooth Caries Alveolar bone Pulpitis Soft tissue Apical infection Facial space
    • Fascial space ---loose connective tissue Among skin, maxillary and muscle •Purulent--- spreading way •Do not exist in healthy state •Become filling during infection
    • Blood and lymphoid system Beneficial Abundance Harmful Cavernous sinus thrombosis Encephalic infection Dangerous triangle •Lacking valves •Cavernous sinus
    • Micro Flora   Varied Numerous • Sterile • Simple • Complex     Streptococcus hemolyticus Staphylococcus aureus Escherichia coli Anaerobe
    •  Odontogenic infection  Adenogenous infection  Traumatic infection  Hematogenous infection  Iatrogenic infection
    • • Periapical infection • Pericoronitis
    • Fistular Cellulitis Intraoral soft tissue abscess Septicemia Acute-chronic Periapical infection Deep fascial space infection Osteomyelitis Ascending facialcerebral infection
    • Lower third molar
    •  Local Signs and Symptoms  Systemical Signs and Symptoms
    • Locally Pain  Swelling  Surface erythema  Pus formation  Limitation of motion 
    •      Fever Lymphadenopathy Malaise Toxic appearance Elevated white blood cell count
    • Acute stage  Chronic stage 
    • Host  Antibiotic therapy  Surgical drainage and incision 
    • Defense Mechanisms   Migration of white blood cell Production of antibodies Critical Principles   Most important factor --- final outcome The infections--- cured by the host, not by antibiotics
    • Antibiotic era causative organism  sensitivity  specific, narrow-spectrum antibiotic  least toxic antibiotic  drug history(success, allergic and toxic) 
    • Proper dose  Proper time interval  Proper route of administration(oral, parenteral)  Combination antibiotic therapy 
    • How to judge the pus formation?  Purposes of surgical drainage and incision  Principles of surgical drainage and incision 
    • Characteristic Three stages Inoculation     Cellulitis Abscess   Duration--- >5 days Palpation---Fluctuant Appearance---Reddened Needle aspiration B-ultrasound CT
    • Rid the body of toxic purulent material  Decompress the tissues  Allowing better perfusion of blood containing antibiotics and defensive elements  Increased oxygenation of the infected area 
    • Ludwig’s angina
    • Place the incision in an esthetically acceptable  Place the incision in a dependent position to encourage drainage by gravity  Dissect bluntly through deeper tissues and explore all portions of the abscess  Place a drain and stabilize it with sutures 
    • Principles of surgical drainage & incision
    • Chronic stage Surgical removal of the focus  Lesion tooth---Impacted tooth  Osteomyelitis
    • Infection in oral & maxillo-facial region The trait of Infection  Odontogenic infection  mutation of infectious  Diagnosis of the infection  Diagnosis of the abscess formation  The principles of the treatment 