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Academia edu cancer short comments 1. Document Transcript

  • 1. 0 • Home • Analytics • EFRUZ ASİLOĞLU • • Inbox • Find Friends • Invite • Account Settings • Edit My Webpage • Log OutNotificationsView All Notifications Search PeopleType to search People, Research Interests and UniversitiesSearching...EFRUZ ASİLOĞLU"EFRUZHU CANCER(CARCİNOGENESİS)THEORY":1.İNADEQUATE=İNEFFİCİENCY ENERGY2.CLONED=MUTATİON OF NORMAL CELL DNA SEQUENCE=ABERRANTSİGNALİNG PATHWAYS MUTAT... moreabout a month agoediteditAboutIndependent ResearcherPrimary Research Interest:
  • 2. MOLECULAR CANCER BİOCHEMİSTRYRecent UpdatesEFRUZadded 5 books a day agoEFRUZadded 21 books a day agoEFRUZ added a talk, a teaching document. a day ago editBooks (25) MOLECULAR CARCİNOGENESİS VOİCED BOOKS 5.1. VİDEO CANCER BOOKS 1-2.1 MOLECULAR CARCİNOGENESİS VOİCED BOOKS 5.2. MOLECULAR CARCİNOGENESİS VOİCED BOOKS 2.2.editPapers (32) "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 24. "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 25 "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 17. "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" PAPERS 2.editTalks (5) "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" You Tube video link 1 "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" COMMENTS 4. "EFRUZHU CANCER (CARCİNOGENESİS)THEORY"COMMENTS 1. "EFRUZHU CANCER (CARCİNOGENESİS) THEORY"COMMENT 3.editTeaching Documents (10) "EFRUZHU PRİNCİPLES OF CARCİNOGENESİS (CANCER) THEORY" "EFRUZHU CANCER (CARCİNOGENESİS) THEORY" DOCUMENTSeditResearch Interests (8)Primary • MOLECULAR CANCER BİOCHEMİSTRY • MOLECULAR CANCER ENDOCRİNOLOGY • MOLECULAR CANCER BİOLOGY • MOLECULAR CANCER GENETİCSview all (8) Status Updates (50)editWebsites (9)AOLBLOGGERFACEBOOKGMAİLShow All (9) Add MoreStatus Updates
  • 3. "EFRUZHU CANCER(CARCİNOGENESİS)THEORY":1.İNADEQUATE=İNEFFİCİENCY ENERGY2.CLONED=MUTATİON OF NORMAL CELL DNA SEQUENCE=ABERRANTSİGNALİNG PATHWAYS MUTATİONAL RECEPTORS. 3.CHRONİC İNFAMMATİONAND CONTİNUOUS SİGNALİNG MOLECULES FROM OUTSİDE.4.NORMAL=SELFDNA SEQUENCE SUPPESSİON=REPRESSİON DİFFERENT DEGREE=MALİGNİTYDEGREE.CARCİNOGENESİS İN SUMMARY ABOVE.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 50. WİLL READİNG PLEASE. about a month agodelete EFRUZ ASİLOĞLU THE FİRST AND THE SECOND RULES CHANGEABLELİNE(ROW,TURN) EACH OTHER. about a month ago Add Comment "THE MOST İMPORTANT DİSCOVERY EFRUZHU CANCER(CARCİNOGENESİS)THEORY"about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 49. about a month ago Add Comment "EFRUZHU CANCER(CARCİNOGENESİS)THEORY" TO RESEARCH HAD BEENBEGUN FİRST METAPHYSİCAL SCİENCES MACRO LEVEL AND THEN İTSEQUALİZE POSİTİVE SCİENCES MOLECULAR LEVEL.BOTH OF THESE AREOPPOSİTE/CONTRARY LİKE CANCER CELL: DUAL ROLE/DUAL ACTİON/BİNARYİDENTİTY İF TWO OPPOSİTE/ SYMETRİC WORKS TOGETHER SUİTABLEYİELD/PRODUCTİON POSSİBLE.THANK YOU VERY MUCH ALL OF THE WORLDCİTİZENS...about a month agodeleteComment | Like
  • 4. delete EFRUZ ASİLOĞLU 48. about a month ago Add Comment DUAL FUNCTİON/DUAL ROLE AT HİSTON PROTEİNS NOT ENOUGHENERGY=ATP THAT COULD OPPENED SO SELF DNA SEQUENCE NOTTRANSCRİPTİON BUT GLYCOLYSİS DOMİNANTLY USES ABERRANT SİGNALİNGPATHWAYS AND FOREİGN= NONSELF DNA SEQUENCE THAT COULD OPPENEDTRANSCRİPTİON EXPRESSİON İS HAPPENED.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 47. about a month ago Add Comment NİTRİC OXİDE (NO) VERY İMPORTANT MOLECULES THAT FREE RADİCAL TOİNHİBİT THE MİTHOCHONDRİON AND AS MESSENGER MAKESHYPERMETHYLATİON OF SELF=NORMAL DNA SEQUENCESUPPRESSİON=REPRESSİON=SİLENCE İN THE NUCLEUS.BUT SAME TİMENONSELF=MUTATİONS=ACTİVATİON TRANSCRİPTİON EXPRESSİON(ABERRANT SİGNALİNG).OTHER NORMAL-SELF SİGNALİNG PATHWAYS NEEDATP=ENERGY THAT AUTOREGULATİON=FEEDBACK MECHANİSM COULD NOTWORK AS PYSİOLOGİC.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 46. about a month ago Add Comment PATHWAYS,UPREGULATİON OF NORMAL CELL RECEPTOR.TWO SCİENCESCAN BE PRODUCE THİS DRUG. İF WE HAVE REMEMBER CANCER CELL HASBİNARY İDENTİTY SO SOLUTİON İS POSSİBLE SAMEPARALLEL.GENETİCS(İNCLUDE MOLECULAR BİOLOGY)
  • 5. +BİOCHEMİSTRY.BECAUSE CANCER CELL PROBLEMS TO BELONGMOLECULAR GENETİCS AND BİOCHEMİCAL.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 45. about a month ago Add Comment I WOULD LİKE TO MAKE SUGGESTİONS:MUST BE RESQUE THE CANCERCELL İTS MUTATİON WİTH COMPLEMENTARY NORMAL DNASEQENCE,REMOVE/DECREASE ABERRANT SİGNALİNG PATHWAYS,DOWNREGULATİON OF MUTATİONAL RECEPTOR ,HİGH ENERGY PHOPHATEATP,ANTİ-İNFLAMATİON,DECREASE SİGNALİNG MOLECULE FROM OUTSİDEADİTİONAL NORMAL DNA SEQUENCE İNCREASE HİSTONMODİFİCATİON,DECREASE HYPERMETHYLATİON=SİLENCE,İNCREASİNGNORMAL SİGNALİNGabout a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 44. about a month ago Add Comment CANCER CELL WANTS TO HELP COULD PASSED TO NORMAL CELLCONDİTİON BASİS OF HOMEOSTATİC PHYSİOLOGY.CHEMOTHERAPY/RADİATİONTHERAPY İS NOT REAL-SUİTABLE OF CANCERCELL REALİTY/PRACTİCALLY.ANY LİVİNG ORGANİSM DOES NOT ACCEPT TODİE AND WAR STİMULATE TO WAR/TO FİGHT AT THE END THİS RELATİONTURNS VİCİOUS CİRCLE.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 43. about a month ago
  • 6. Add Comment EFRUZHU CANCER(CARCİNOGENESİS)THEORY İS REALLY TRUE=100%PERCENT BUT İMPOSSİBLE TO PROVE DİRECTLY AND DETAİLED.ANY STAGEOF CANCER DİSEASE CAN SOLVE TOTALLY İF THİS THEORY TO TAKE İNTOCONSİDERATİON CANCER CELL WİLL HAVE DİFFERENCİATED AND BECOMEMATURE CELL.THİS THEORY TO EXPLAİN MULTİCOMPLEX MOLECULARRELATİONSHİP LİKE UNİVERSE İN SUMMERY. THATS ENOUGH FORSOLUTİON.WE WOULD LİKE REAL NEW MOLECULAR SOLUTİON THAT ...about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 42. about a month ago Add Comment CANCER CELL LİVİNG STYLE İS İRREGULAR=LAWLESS DEPENDENCE OFMUTATİONAL DNA SEQUENCES,ABERRANT SİGNALİNGPATHWAYS,MUTATİONAL RECEPTORS,CONTİNUOS SİGNALİNG FROMOUTSİDE.ALSO CANCER CELL PARASİTİC GLUCOSE CONSUMER CHARACTERRELATİON SAME FACTORS.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 41. about a month ago Add Comment CARCİNOGENESİS DEPENDENCE LOST OF OXİDANCE(FREERADİCAL)/ANTİOXİDANCE BALANCE,THAT CAUSE OF DNAMUTATİONS,BREAK DOWN(SUPPRESSİON=İNHİBİTİNG)ENERGYGENERATİON/PRODUCTİON,CHRONİCAL İNFLAMMATORY CONDİTİON ANDCHAOTİC RELATİONSHİP VERY İMPOTANT POİNTS.about a month agodeleteComment | Like
  • 7. delete EFRUZ ASİLOĞLU 40. about a month ago Add Comment SİNGLE CANCER CELL AT FİRST HAVE BEEN CHANGED İTS İDENTİTY BYENVİRONMENTAL CELLS THAT KEEP İTS LİFE(PREVENT İTS LİVİNG),SELFRENEWAL MEANS CULTURAL ADAPTATİON(TO ACCEPT,TO APPROPRİATE BYENVİRONMENT=CULTURE).BUT" CANCER CELL LİVİNG İS UNDER SUBOPTİMALLİVİNG CONDİTİONS".about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 39. about a month ago Add Comment CANCER CELL HAS A LOT OF OPPOSİTE DYNAMİCS ALL OF THESE ARE NOTCONTRADİCTİON,THİS İS SHOW US İF LİFE POSSİBLE TO MANAGESYMMETRİCAL WOULD BECOME BALANCED.DUAL ROLE/DUAL ACTİONAPPROXİMATELY WHOLE CHARACTER OF CANCER CELL BUT ABERRANTSİGNALİNG CAUSE OF EXPRESSİON FOREİGN=MUTATİON DNA PERSONDOMİNANTLY AND SELF=NORMAL DNA COULD NOT EXPRESSİON=SİLENCE İNTHE LİFE(NOT ACTİON).about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 38. about a month ago Add Comment CANCER İS A DİVİNE/HEAVENLY DİSEASE THAT MEANS ALL OFOPPOSİTES/CONTRARİES COULD SELF EXPRESSİON PROVİDE TO BE ATPEACE,RİCH,PRODUCTİVE LİFE İS POSSİBLE İN THE WORLD.about a month agodelete
  • 8. Comment | Likedelete EFRUZ ASİLOĞLU 37. about a month ago Add Comment "EFRUZHU CANCER (CARCİNOGENESİS)THEORY":CANCER CELL HAS DUALROLE 1-ONE SİDE WANTS TO DİE THAT NORMAL DNA SEQUENCE AND İTSDİNAMİCS ARE MANAGİNG 2-OTHER SİDE WANTS TO LİVEFOREVER(İMMORTAL)THAT MUTATİONAL SEQUENCE AND DYNAMİCS AREMANAGİNG.CANCER CELL ALL STEPS DUAL ROLE-DUAL FUNCTİONSDEPENDENCE OF BİNARY İDENTİTY.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 36. about a month ago Add Comment POPULATİON AGAİNST NATURE OF CANCER CELL.LİVİNG CELL JUST LİKEHUMAN THAT LİVİNG İS DİFFİCULT TO BE OBLİGATİON TO EMİGRATEANOTHER SUİTABLE LİVEABLE AREA.CANCER CELL DOES NOT WANT TO DİEAND İT İS LOOKİG FOR SOURCE AREA AS RİCH NUTRİENT AND OXYGEN.LATEROF METASTASİS TO ARRİVE CANCER CELL CONSUME OXYGEN AND NUTRİENTAS PARASİT, CHAOS AND İNFLAMMATİON ETC LOCAL OTHER CELL HAVEBEEN DONE NEOPLASTİC TRANSFORMATİON .about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 35. about a month ago Add Comment WHY CANCER CELL WANTS TO MAKE METASTASİS FİRST CARCİNOGENESİSSTARTİNG İN ONE CELL LATER LOOK LİKE ONE ANOTHER TO İNCREASE-TOMULTİPLY AT THE END. COLONY WİLL HAVE HAPPENED, CANCER CELL
  • 9. PERSONAL CHARACTER İS PARASİTİC SO EVERY PERSON(LOCALLY)WİLLBECOME İN THE COLONY NEOPLASTİC PARASİT. WHİCH PERSON WİLL WORKSAS SOURCE(NUTRİENT AND OXİGEN).SO İT HAS TO EMMİGRATE DİFFERENTLİVEABLE AREA BECAUSE SAME CHARACTER PERSON-about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 34. about a month ago Add Comment ALL CANCER (CARCİNOGENESİS)STARTİNG POİNT 1-NORMAL CELL DNASEQUENCE İS CLONED(BY VİRUS,İMMUN CELLS)2-İNADEQUATE ENERGY 3-CHRONİC İNFLAMMATİON(OXİDANCE-OXİDATİVE STRESS),İNADEQUATENUTRİENT AND HYPOXİA.EXCESS FAT,PROTEİN,KARBONHİDRATE (BREAKDOWN ENERGY METABOLİSM)EXCESS SİGNALİNG MOLECULE(HORMONES)CAUSE OF LOSS THE CELL AUTOCONTROL(BY İTSELF).about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 33. about a month ago Add Comment ONCES NORMAL CELL TRANSFORM TO CANCER CELL THAT İT HASDİFFERENT CELL İDENTİTY=MUTATİON LATER, TO BE ATTRACT ATTENTİONWHEN CANCER CELL MAKE BEHAVİOUR-ACTİON,THE OTHER NORMAL CELLS(NEAR AREA)HAVE MADE MİMİCS=LOOK LİKE=COPY OF CANCERCELL,BECAUSE THEY HAVE SAME İDENTİTY=CANCER CELL MUTATİONS.SOLOOK LİKE=MİMİCS=İMİTATİON, VİA SİGNALİNG WOULD HAVE BEENHAPPENED AUTOMATİCALLY.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 32. about a month ago
  • 10. Add Comment "BASİC İDEA OF EFRUZHU CARCİNOGENESİS THEORY ":FİRSTLYİNADEQATE ENERGY OF NORMAL CELL,"NONSELF ANTİGENİC DYNAMİCS=SİNGLE CELL=ONLY ONE CELL-REALLY NOT FOREİGN",DESTRUCTİVEPRODUCTS ETC MAKİNG TO BEGİN PHAGOSİTOSİS BY İMMUN CELLS SOSCENARİO İS STARTİNG.about a month agodeleteComment | Likedelete EFRUZ ASİLOĞLU 31. about a month ago Add Comment ← Previous 1 2 3 Next →Want an instant answer to your question? Check the FAQs. Send • Job Board • About • Press • Blog • Were hiring engineers! • FAQ • Feedback • Terms • Privacy • Copyright Academia © 2012