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Congenital lesions of larynx
1. CONGENITAL LESIONS OF
LARYNX AND STRIDOR
DEPT OF OTORHINOLARYNGOLOGY
JJM M C
DAVANAGERE
2. LARYNGOMALACIA
• Most common congenital abnormality of larynx
• Condition manifests at birth or soon after and
usually disappears by two years of age
• Characterized by excessive flaccidity of
Supraglottic larynx which is sucked in during
inspiration producing stridor and cyanosis
• Stridor subsides on placing child in prone position
and increases with crying
• Direct laryngoscopy elongated and curled up
epiglottis (omega shaped), floppy AE folds and
prominent arytenoids
• Treatment is conservative
• In severe stridor tracheostomy
3.
4. CONGENITAL VOCAL FOLD
PARALYSIS
• Results from birth trauma when
RLN is stretched during breach or
forceps delivery
• Can also results from anomalies of
CNS
5. CONGENITAL SUBGLOTTIC
STENOSIS
• Due to abnormal thickening of cricoid
cartilage or fibrous tissue seen below
the vocal cords
• Child may remain asymptomatic till
URTI causes dyspnoea and stridor
• Diagnosis subglottic diameter less
than 4mm in full term neonate (normal
4.5-5.5mm) or 3mm in premature
neonate (normal 3.5mm)
• Most cases improve as larynx grows but
some may require surgery
6.
7. LARYNGEAL WEB
• Due to incomplete recanalization of
larynx
• Seen between the vocal cords with
concave posterior margin
• Presents as airway obstruction, weak
cry or aphonia dating from birth
• Treatment depends on thickness of web
• Thin web cut with knife or CO2 laser
• Thick web excision via laryngofissure
and placement of silicone keel and
subsequent dilatation
8.
9. SUBGLOTTIC HAEMANGIOMA
• Though congenital patient is asymptomatic till 3-6 months
of age
• About 50% have associated cutaneous haemangioma
• May present with stridor which increases on agitation and
crying due to venous filling
• Direct laryngoscopy shows reddish blue mass below
vocal cords
• Biopsy is sometimes, not always associated with
hemorrhage
• Treatment depends on individual case
Tracheostomy and observation
Steroid therapy
CO2 laser excision
11. LARYNGO-OESOPHAGEAL
CLEFT
• Due to failure of fusion of cricoid
lamina
• Presents with repeated aspiration
and pneumonitis
• Coughing, choking and cyanosis
are present at the time of feeding
• Surgically treated
12. LARYNGEAL CYST
• Arises in the AE folds and appears as
bluish fluid filled smooth swelling in
Supraglottic larynx
• Respiratory obstruction tracheostomy
• Needle aspiration or incision and
drainage provides emergency airway
• Treatment is deroofing the cyst or
excision with CO2 laser
13.
14. STRIDOR
• Defined as noisy respiration
produced by turbulent airflow
through narrowed air passage
• Classified as Inspiratory, expiratory
and biphasic
15. STRIDOR
• Inspiratory stridor produced in
obstructive lesion of Supraglottic or
pharynx
• Expiratory stridor lesions of
thoracic trachea, primary and
secondary bronchi
• Biphasic lesions of glottis,
subglottis and cervical trachea
18. STRIDOR-CAUSES
• Trachea and bronchi:
Congenital: atresia, stenosis, malacia
Inflammatory: tracheobronchitis
Neoplastic: tumors
Traumatic: foreign body and stenosis
• Lesions outside respiratory tract
Congenital: vascular rings, oesophageal atresia, tracheo-
oesophageal fistula, cystic hygroma
Inflammatory: retroeosophageal abscess
Traumatic: foreign body oesophagus
Tumors: masses in the neck
19. STRIDOR-MANAGEMENT
• History: onset, progression, duration,
relation to feeding, cyanotic spell,
aspiration or ingestion of foreign body,
laryngeal trauma
• Physical examination: signs of
respiratory distress, stridor with phase
of respiration, associated features like
fever, wheeze, snoring, hoarseness,
muffled voice
20. STRIDOR-MANAGEMENT
• Investigations
X-ray chest and soft tissue neck AP and
lateral view
Fluoroscopy
CT of neck and chest
Oesophagogram
Angiography
Xeroradiography
Direct laryngoscopy
Pan endoscopy