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“ It is the protection of a pulp exposed by traumatic fracture or in the course of excavating deep dentinal caries. Protection is provided by placing a medicated or nonmedicated material in direct contact with the pulp tissues to promote a reparative reaction.”
Weiss, 1970 -healthy pulp exist beneath DPC even without dentin bridge.
Stanley, 1989 –Size of exposure doesn’t matter. For healing : Calcium hydroxide dressing should be in contact with living tissues to stimulate odontoblastic regeneration. Brannstrom 1976- placed filter paper soaked with S. sanguis for 2 days &10 weeks later found thick dentinal bridge.
Ricketts 2001 –with increased age fibrous pulp tissue increase and blood supply decrease. So, the capacity to respond to DPC.
Stanley, 1972 – In cervical cavity, no pulp tissue should be present coronal to the exposure or it will lead to reactionary dentin formation which would further restrict blood supply leading to necrosis & failure.
With creation of microfissures b/w calcium hydroxide & dentin, exudate of pulpal fluids produced .Its due to outward hydraulic pressure. Which leads to dissolution of bacterial invasion pulp inflammation pain.
Cox et al 1996 –CH induced dentin bridges contained multiple defects, porosities leading to bacterial & fluid microleakage.
Acc. To Schuers 2000 – Pulp capping with resin based adhesive composite systems are at the moment the only realistic alternative to CH products.
Martin et al 2002- teeth covered with CH with IRM were superior to other teeth capped with adhesive composite system. IRM seal resulted in significantly better healing emphasizing critical importance of good coronal seal after pulp capping.
Direct pulp capping is a procedure used in asymptomatic teeth with deep caries reaching upto pulp. It is another method than Indirect pulp capping to treat deep caries but it is not a preferred method in children as success rate is very low, like indirect pulp capping in this also a suitable medicament is placed to induce dentin bridge formation.