• Save
Upcoming SlideShare
Loading in...5







Total Views
Views on SlideShare
Embed Views



0 Embeds 0

No embeds



Upload Details

Uploaded via as Microsoft PowerPoint

Usage Rights

© All Rights Reserved

Report content

Flagged as inappropriate Flag as inappropriate
Flag as inappropriate

Select your reason for flagging this presentation as inappropriate.

  • Full Name Full Name Comment goes here.
    Are you sure you want to
    Your message goes here
Post Comment
Edit your comment

Stroke Stroke Presentation Transcript

  • DEFINATION Stroke or brain attack is the sudden loss of neurological function caused by an interruption of the blood flow to the brain. Classification:  Ischemic stroke : is the most common type results when a clot blocks or impairs blood flow, depriving the brain of essential oxygen and nutrients.  Hemorrhagic stroke: occurs when blood vessels rupture, causing leakage of blood in or around the brain.
  • EPIDEMIOLOGY • Third leading cause of death • Most common cause of disability among adults in U.S • The incidence is about 1.25 times greater for males then females M>F • Compared to whites, African-Americans have twice the risk of first ever stroke
  • ETIOLOGY • • • • • • • • Atherosclerosis Ischemic strokes Cerebral thrombosis Cerebral embolus Intracerebral hemorrhage Cerebral hemorrhage Subarachnoid hemrrohage Arteriovenous malformation (AVM)
  • RISK FACTORS Modifiable: • History of TIAs • HTN • Atrial fibrillation • High RBC count • Undesirable levels of blood cholestrol • Heart disease • Cigarette smoking • Alcohol consumption • Obesity • DM
  • Non-modifiable: • • • • • Prior stroke Age Race Gender Family history of stroke
  • PATHOPHYSIOLOGY Interruption of blod flow Complete cerebral circulatory arrest Irreversible cellular damage with core area of Ischemic Ischemic focal infarction cascade penumbra Release of excess neurotransmitters (glutamate and aspartate) Damage brain cells
  • TRANSIENT ISCHEMIC ATTACK (TIA) • Temporary interruption of blood supply to the brain. • Symptoms last for only a few minutes or for several hours • Not last longer then 24 hours • No evidence of residual brain damage or permanent neurological dysfunction • Etiological factors: occlusive episodes, emboli, reduced cerebral perfusion(arrhythmias, decreased cardiac output, hypotension, overmedication with antihypertensive medication or cerebrovascular spasm
  • WARNING SIGNS OF STROKE: • Sudden numbness or weakness of the face, arm or leg esp on one side of the body • Sudden confusion, trouble speaking or understanding • Sudden trouble seeing in one or both eyes • Sudden trouble walking, dizziness, loss of balance or coordination • Sudden, severe headaches with no known cause
  • ACA syndrome ACA is the smaller terminal branch of the internal carotid artery. • Supplies: • Medial aspect of cerebral hemisphere(frontal & parietal lobes) • Sub cortical structures BG ( anterior internal capsule, inferior caudate nucleus, anterior 4/5th of corpuscallosum)
  • Signs and symptoms : • • • • Contra lateral hemi paresis (LE > UE) Contra lateral hemi sensory loss (LE > UE ) Urinary incontinence Problems with imitation and bimanual tasks, apraxia Structures involved : • Primary motor area, medial aspect of cortex, internal capsule • primary sensory area • Posteriomedial aspect of superior frontal gyrus • corpus callosum
  • MCA syndrome MCA is the second of the two main branches of the internal carotid artery. Supplies: • Lateral aspect of cerebral hemisphere( frontal, temporal, and parietal lobes ) • Subcortical structures (IC, corona radiata , globus pallidus, putamen )
  • Signs and symptoms: • • • • • • Contralateral hemiparesis (UL and face > LL) Contralateral sensory loss (UL and face > LL) Motor speech impairment Receptive speech impairement Global aphasia Perceptual deficits (unilateral neglect, anosognosia, apraxia and spatial disorganization) • Contralateral homonymous hemianopsia • Sensory ataxia Structures involved : Primary motor area, primary sensory area, broca’s area, Wernicke’s area, optic radiation in internal capsule
  • PCA syndrome PCA is the terminal branches of the basilar artery. Supplies: • Occipital lobe • Medial & inferior temporal lobe • Upper brainstem • Most of thalamus
  • Signs and symptoms:  Peripheral territory: • Contralateral homonymous hemianopsia • Visual object agnosia • Prosopagnosia • Dyslexia • Memory defect  Central territory: • Post stroke (thalamic ) pain • Sensory impairments • Involuntary movements, intention tremor, hemiballismus • Contralateral hemiplegia • weber’s syndrome ( 3RD N. PALSY + CONTRALATERAL HEMIPLEGIA)
  • Vertebrobasilar artery syndrome 1. Medial medullary syndrome( Dejerine syndrome) Signs and symptoms • Ipsilateral: paralysis with atrophy of half tongue with deviation to the paralyzed side when tongue is protruded • Contralateral:  Paralysis of UE and LE  Impaired tactile & proprioceptive sense(medial lemniscus) Structures involved CN XII, corticospinal tract or medial lemniscus
  • 2. Lateral medullary Syndrome (Wallenberg’s syndrome): Signs and symptoms •     Ipsilateral: Decreased pain & temp sensation over face(V n.) Ataxia of limbs and gait Vertigo, nausea, vomitting, nystagmus Horner’s syndrome: miosis, ptosis, decreased sweating(sympathetic tract)  Dysphagia, hoarsness of voice, paralysis of vocal cord(IX, X n.)  Sensory impairment of ipsilateral UE, trunk, or LE • Contralateral : Impaired pain and thermal sense over 50% of body Structures involved CN V, Cerebellum, vestibular nuclei, descending sympathetic tract, CN IX
  • 3. Complete basilar artery syndrome ( locked-in syndrome ) Signs and symptoms • Tetraplegia • Bilateral cranial nerve palsy • Coma • Cognition is spared Structures involved • Corticospinal tracts bilaterally • Long tracts to cranial nerve nuclei bilaterally • Reticular activating system
  • 4.Medial inferior pontine syndrome: Signs and symptoms • Ipsilateral:  Nystagmus  Ataxia of limb & gait  Diplopia on lateral gaze(6th n.)  Paralysis of conjugate gaze to side of lesion • Contralateral:  Paresis of face, UE and LE  Impaired tactile & proprioceptive sense over 50 % of the body Structures involved  Vestibular connections, middle cerebellar peduncle, CN VI, corticobulbar and corticospinal tract
  • 5.Lateral inferior pontine syndrome: Signs and symptoms • Ipsilateral:  Nystagmus,vertigo,nausea,vomitting (vest. N.)  Facial paralysis(7th n.)  Ataxia(cerebellar peduncle)  Impaired sensation over face(5th n.) • Contralateral:  Impaired pain & thermal sense over half the body(spinothalamic tract)
  • 6. Medial midpontine syndrome Signs and symptoms • Ipsilateral:  Ataxia of limb & gait (middle cerebellar peduncle) • Contralateral:  Paralysis of face,arm,leg(CST, corticobulbar tract)  Deviation of eyes 7. Lateral midpontine syndrome Signs and symptoms • Ipsilateral:  Ataxia of limbs( middle cerebellar peduncle)  Paralysis of muscles of mastication( CN V)  Impaired sensation over face(CN V )
  • 8. Lateral superior pontine syndrome Signs and symptoms • Ipsilateral:  Ataxia of limb & gait(middle cerebellar peduncle)  Dizziness, nausea, vomiting, nystagmus( vest nuc)  Horner’s syndrome( desc symp tract) • Contralateral:  Impaired pain, thermal sense (spinothalamic tract)  Impaired touch, vibration & position sense(medial lemniscus) 9.Medial superior pontine syndrome Signs and symptoms  Cerebellar ataxia( Sup and midd cerebellar peduncle )  Paralysis of face, UE and LE( CST & CBT)
  • PRIMARY IMPAIRMENTS 1. • • • • • SENSATION Rarely absent Impaired Most common distribution pattern: face-UL-LL Proprioceptive losses common Loss of superficial touch and pain and temp sensation common • Abnormal sensations such as numbness, dysesthesias, or hyperesthesia • Profound hemisensory loss
  • 2. PAIN • Ischemic stroke or hemorrhagic stroke result severe headache and face pain. • Post stroke thalamic pain characterised by : • Constant, severe burning pain with intermittent sharp pains. • Paroxysmal spasms of pain may be triggered by simply stroking , pin prick, heat or cold 3. • • • VISUAL CHANGES Homonymous hemianopsia Visual neglect Forced gaze deviation
  • 4. MOTOR FUNCTION Stages of motor recovery
  • 5. ALTERATIONS IN TONE FLACCIDITY SPASTICITY • Spasticity stronger in: Upper limb • Scapular retractors • Shoulder adductors,depressors,internal rotators • Elbow flexors, forearm pronators • Wrist & finger flexors Lower limb • Pelvic retractors • Hip adductors, Internal rotators • Knee extensors • Ankle plantar flexors • Toe flexors
  • 7. ABNORMAL REFLEXES • Hyporeflexia with flaccidity & hyperreflexia with spasticity • Primitive reflexes • Associated reactions 8. ALTERED COORDINATION 9. ALTERED MOTOR PROGRAMMING 10. POSTURAL CONTROL AND BALANCE • Ipsilateral pushing ( pusher syndrome ) Is an unusal motor behaviour characterised by active pushing with the stronger extremities toward the hemiparetic side, leading to a lateral postural imbalance
  • 11. Cognitive & perceptual disorders: • Body scheme & body image disorders • Spatial relation disorders figure-ground dis • Agnosias form dis • Apraxias • anosognosia • Hemineglect • Memory, attention disorders • Confabulations • Dementia • Delirium
  • 12.Speech, language and swallowing • Aphasia • Fluent and non fluent aphasia • Global aphasia • Dysarthria • Dysphagia 13. Emotional status • Pseudobulbar affect • Apathy • Euphoria • Depression 14. Bladder and bowel dysfunction • Urinary incontinence
  • COMPLICATIONS AND INDIRECT IMPAIRMENTS  Musculoskeletal • Contractures • Disuse atrophy • Muscle weakness • Osteoporosis  Neurological • Seizures • Hydrocephalus  Cardiovascular / Pulmonary • Thrombophlebitis / Deep vein thrombosis • Cardiac function : impaired CO and serious rhythm disorders • Pulmonary function : decreased lung volume, decreased pulmonary perfusion and vital capacity