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  • What controls or regulates the fluids in our body? Thirst –simplest way to maintain fluid balance Thirst center failure- onconscious or confused pt. To not respond Which age group is most prone to dehydration because their body’s weight is mostly water?
  • What also is increased here?
  • Increased risk for fluid/electrolyte imbalance with decreased muscle since muscle cells hold more water
  • NOTE: Potter & Perry speaks to the “percentage of body weight” 40% of BODY WEIGHT = ICF fluid 20% of BODY WEIGHT = ECF fluid Transcellular fluid is a negligible amount
  • This is reverse of adults THEREFORE the infant is more susceptible to fluid loss
  • SEE NEXT SLIDES FOR IN-DEPTH
  • Water is a solvent Concentration of particles in solution (pulling action = osmolarity) Isotonic have almost same osmolarity as plasma therefore there is no pull
  • Osmosis, by the way, is the reason that drinking salt water will kill you. The HIGH osmolarity salt water in the GI system rapidly pulls water into the GI system and excretion – rapidly dehydrating cells SEE NEXT SLIDES FOR FURTHER DISCUSSION
  • Used for post op, decreases intracellular edema, fosters normal BP and good urinary output. D51/2NS, D5NS, D5RL Hyperal
  • ECF- extracellular fluids
  • ICF intracellular fluid - fluid inside the cell D5W isotonic / Normal saline solution is isotonic because it has almost the same concentration of sodium as blood. Used to replace Ecvlume
  • Hypertonic
  • EDEMA
  • Filtration- from pressure to low pressure
  • Dehydration : Fluid intake is not sufficient to meet the body’s needs. Dehydration- if water isn’t adequately replaced dehydration results Dx Tests Elevated HCT Elevated NA Sp. Gravity above 1.030 Monitor lab work Cause- unless unconscious Sudden wt. change is a major indicator of fluid loss
  • Oral- keep fluids at bedside, offer frequently IV fluids, blood & other parenteral measures Hyperal etc. Meds- depending on the cause Diarrhea give anti diarrhea meds Vomiting give anti emetics Vasopressors if pt. In shock cause vasoconstriction and increase BP
  • Increase in vascular blood Third spacing could be in the abd- ascites pleural effusion in the lungs
  • Retention- Intake- Poorly controlled IV therapy/ rapid hypertonic solution/ excessive sodium bicarb / excessive Na intake
  • Drug therapy- - diuretics for overhydration increases excretion of water and sodium Diet-- restricting fluid and sodium intake Monitor lab work
  • 1 mEq MILLIEQUIVALENT = 1 MG OF HYDROGEN
  • Each will be discussed except Bicarbonate as that plays a role in acid base balance which will be covered in NR33
  • Na concentrations effected by water intake and salt untake Hormones -Aldsterone
  • Causes Poor IV therapy- IV therapy increased water in blood Na is diluted CHF Renal Failure GI: vomiting diarrhea drainage Skin: sweating burns diuretic drugs TX Diet- foods high in sodium - IV solutions ordered if hypovolemia (low volume) Fluid excess- osmotic diuretics ordered to promote excretion of water rather than sodium (mannitol) Fluid restriction till Na returns to norm Lop diueretics to to remove excess fluid Assess: VS skin integrity, seizures, I & O/ monitor lytes
  • Causes- increased Na intake- rapid infusion of saline solution/po intake loss of water – diarrhea/DM/decreased water intake/ impaired thirst center/can’t swallow Fluid shift from ICF to ECF ….(Na pulls h2o out of cells, kidneys excrete Na and water follows) Tx-if caused by fluid loss Need slow gradual return to normal Na+ by IV hypotonic solution 0.45%NS Pt. Teaching avoid high Na foods, canned soups, processed foods, ketchup AVOID antacids high in sodium bicarb I&O, review diet, meds, Moniotr weight, note change LOC
  • Effects skeletal/cardiac/smooth muscle Causes: Inadequate intake Alcoholism/ Diuretics Excessive Vomiting & diarrhea Tx ID cause High K diet, …oranges, broccoli, meat protein foods,banana, apricots PO supplements common IV therapy always diluted…
  • … (false rise due to tight tourniquet or hemolized specimen) occurs Poor elimination by kidneys Parathesia -tingling Tx-Depends on cause Hold Kmeds, low K diet orderd Kayexalate administered to increase excretion of K IV therapy add volume to dilute K+ Monitor for fluid overload.
  • 8.5-10.5mg/deciliter dL Vit D needed for Ca absorption
  • Common after thyroid surgery Chovstek sign-Tap facial nerve in front of ear= facial spasm Trousseau- carpal spasm after BP cuff inflated due to increased neuromuscular excitability TX -Ca supplements…dietary. Dairy green veg, sardines salmon If severe-IV calcium gluconate
  • Remember it’s in the blood not the bones Causes-high intake TX-Depends on cause encourage mobility,immobilization causes demineralization of bones leading to fractures remove parathyroid tumors encourage fluids to prevent renal calculi Lower Ca by IV therapy causes diuresis encouraging kidney excretion Calcium binding meds given to promote excretion of calcium.
  • Flushing due to peripheral vasodilation Resp. deep shallow and slow
  • Tx: correct cause, diet increase Cl, vomiting reduce it, replacement thru IV therapy… can br given orally ie. Salty broth
  • Tx- treat underlying cause, VS, reorient if confused Kussmals –rapid and deep without pauses above 20/min
  • Tx- vs, assess resp, neuro status IV meds safety
  • Tx: Correct the under lying cause..renal failure, diet, decreased absorption, Iv fluids, vs Diet limit foods
  • Note changes- significant factor

Transcript

  • 1. Fluid & Electrolyte Balance NCM 103 Lecture Prepared by: Marjo S. Malabanan
  • 2. Body Fluids  Water= most important nutrient for life.  Water= primary body fluid. Adult weight is 55-60% water.  Loss of 10% body fluid = 8% weight loss SERIOUS  Loss of 20% body fluid = 15% weight loss FATAL  Fluid gained each day should = fluid lost each day (2 -3L/day average)  What is the minimum output per hour necessary to maintain renal function? 30ml/hr
  • 3. Functions of Body Fluid  Medium for transport  Needed for cellular metabolism  Solvent for electrolytes and other constituents  Helps maintain body temperature  Helps digestion and elimination  Acts as a lubricant
  • 4. Mechanisms of Fluid Gain and Loss Gain  Fluid intake 1500ml  Food intake 1000ml  Oxidation of nutrients 300ml (10ml of H20 per 100 Kcal) Loss  “Sensible” Can be seen. Urine 1500ml Sweat 100ml  “Insensible” Not visible. Skin (evaporation) 500ml Lungs 400ml Feces 200ml
  • 5. Regulation of Fluids  Hypothalmus –thirst receptors (osmoreceptors) continuosly monitor serum osmolarity (concentration). If it rises, thirst mechanism is triggered. +Vasopressin (AKA ADH )– increasing H20 reabsorption  Pituitary regulation- posterior pituitary releases ADH (antidiuretic hormone) in response to increasing serum osmolarity. Causes renal tubules to retain H20.  Thirst is a late sign of water deficit
  • 6. Regulation of Fluids (continued )  Renal regulation- Nephron receptors sense decreased pressure (low osmolarity) and kidney secretes RENIN. Renin – Angiotensin I – Angiotensin II  Angiotensin II causes Na and H20 retention by kidneys AND…..  Stimulates Adrenal Cortex to secrete Aldosterone which causes kidneys to excrete K and retain Na and H20.
  • 7. Consider This….  The Geriatric Client -normal physiological aging results in decreased thirst mechanism decreased # of sweat glands decreased renal function -there also may be decreased mobility and/or cognitive function which impacts their ability to get adequate fluid intake.
  • 8. Variations in Body Fluids  Elderly: Have lower % of total body fluid than younger adults  Women: Have lower % total body fluid than men  WHY DO YOU THINK THIS IS ????? Muscle tissue has more H20 content THAN adipose tissue
  • 9. Fluid Compartments Intracellular fluid (ICF)  Fluid inside the cell  Most (2/3) of the body’s H20 is in the ICF. Extracellular Fluid (ECF)  Fluid outside the cell.  1/3 of body’s H20  More prone to loss  3 types: Interstitial- fluid around/between cells Intravascular- (plasma) fluid in blood vessels Transcellular –CSF, Synovial fluid etc
  • 10. Consider this….  Age variations exist in regards to H20 content of fluid compartments  Infants = 60% of H20 is found in ECF 40% of H20 is found in ICF  What might this mean in regards to fluid loss for an infant? Reverse of adults! Infant MORE PRONE to fluid LOSS!
  • 11. Fluid Balance  Dynamic process  Balance between body fluids and electrolytes  Attraction between ions (electrolytes) and water (fluids) causes fluids to move across membranes and leave their compartments.
  • 12. Solvent (H20) Movement  Cell membranes are semipermeable allowing water to pass through  Osmosis- major way fluids transported Water shifts from low solute concentration to high solute concentration to reach homeostasis (balance).
  • 13. Osmolarity  Concentration of particles in solution  The greater the concentration (Osmolarity) of a solution, the greater the pulling force (Osmotic pressure)  Normal serum (blood) osmolarity = 280-295 mOSM/kg  A solution that has HIGH osmolarity is one that is > serum osmolarity = HYPERTONIC solution  A solution that has LOW osmolarity is one that is < serum osmolarity = HYPOTONIC solution  A solution that has equal osmolarity as serum = ISOTONIC solution
  • 14. Hypertonic Fluids  Hypertonic fluids have a higher concentration of particles (high osmolality) than ICF  This higher osmotic pressure shifts fluid from the cells into the ECF  Therefore Cells placed in a hypertonic solution will shrink
  • 15. Hypertonic Fluids  Used to temporarily treat hypovolemia  Used to expand vascular volume  Fosters normal BP and good urinary output (often used post operatively)  Monitor for hypervolemia ! Not used for renal or cardiac disease.
  • 16. Hypotonic Fluids  Hypotonic fluids have less concentration of particles (low osmolality) than ICF  This low osmotic pressure shifts fluid from ECF into cells  Cells placed in a hypotonic solution will swell
  • 17. Hypotonic Fluids  Used to “dilute” plasma particularly in hypernatremia  Treats cellular dehydration  Do not use for pts with increased ICP risk or third spacing risk
  • 18. Isotonic Fluid  Isotonic fluids have the same concentration of particles (osmolality) as ICF (275-295 mOsm/L)  Osmotic pressure is therefore the same inside & outside the cells  Cells neither shrink nor swell in an isotonic solution, they stay the same
  • 19. Isotonic Fluid  Expands both intracellular and extracellular volume  Used commonly for: excessive vomiting,diarrhea  0.9% Normal saline  D5W  Ringer’s Lactate
  • 20. Other Osmotic Factors  ALBUMIN ( a serum protein )  Albumin in the serum has osmotic properties called colloid pressure  Albumin pulls H20 from the interstitial compartments into the intravascular compartments (serum). Helps to maintain BP.  Persons with low serum albumin levels tend to retain fluid in their interstitial layers. What abnormal assessments might you find in the client with low serum albumin levels? Edema, hypotension
  • 21. Hmmm…….  What type of IV fluid (hypotonic – isotonic – hypertonic) might be of benefit to this client with low albumin levels?
  • 22. Consider this….  When tissue injury occurs, proteins pathologically leak from the intravascular space into the intersititial space. Termed: Third spacing  This explains __________ as a sign of the inflammatory process. EDEMA
  • 23. Solute Movement - Diffusion  Movement of solutes from high concentration to low concentration  It is a PASSIVE movement DOWN the concentration gradiant. (requires no energy)  Many body processes use diffusion. Example: O2 and CO2 exchange  Rate is affected by: concentration gradiant, permeability-surface area-thickness of membranes, and size of particles. (Fick’s Law)
  • 24. Solute Movement –other mechanisms  Active transport- requires energy (ATP) to move from low concentration to high concentration (uphill) Example: Na / K pump  May be enhanced by carrier molecules with binding sites on cell membrane Example: Glucose (Insulin promotes the insertion of binding sites for Glucose on cell membranes).
  • 25. Filtration  Solvent AND solute movement  Passage from an area of High Pressure to an area of Low Pressure Termed: Hydrostatic Pressure  Example: Arterioles have higher pressure than ICF Fluid, oxygen and nutrients move into cells Venules have lower pressure than ICF Fluid, carbon dioxide and wastes move out of cells
  • 26. Definitions  An arteriole is a small diameter blood vessel in the microcirculation that extends and branches out from an artery and leads to capillaries.  A venule is a very small blood vessel in the microcirculation that allows blood to return from the capillary beds to the larger blood vessels called veins.
  • 27. Fluid volume deficit FVD (Hypovolemia)  Loss of both H20 and electrolytes from ECF.  Causes include: Increased output, Hemorrhage, vomiting, diarrhea, burns, OR  Fluid shift out of vascular space ( “third spacing” ) into interstitial spaces
  • 28. Dehydration  Isotonic dehydration = H20 & electrolyte loss in equal amounts; diarrhea and vomiting  Hypertonic dehydration = H20 loss greater than electrolyte loss; excessive perspiration, diabetes insipidus
  • 29. Assessment FVD - Hypovolemia Cardiovascular:  Diminished peripheral pulses; quality 1+(thready)  Decreased BP & orthostatic hypotension  Increased HR  Flat neck & hand veins in dependent position  Elevated Hematocrit (Hct) Gastrointestinal:  Thirst  Decreased motility; diminished bowel sounds, possible constipation
  • 30. Assessment FVD – Hypovolemia (continued) Neuromuscular:  Decreased CNS activity (lethargy to coma)  Possible fever  Skeletal muscle weakness  Hyperactive DTR Renal:  Decreased output  Increased spec grav of urine  Weight loss  Hypernatremia Integumentary:  Dry mouth & skin  Poor turgor (tenting)  Pitting edema  Sunken eyeballs Respiratory:  Increased rate and depth
  • 31. Nursing Diagnosis - FVD  Deficient Fluid Volume R/T loss of GI Fluids via vomiting AEB elevated Hct, dry mucous membranes, decreased output, thirst
  • 32. Planning - FVD  Client will demonstrate fluid balance aeb moist mucous membranes, balanced I & O measurements, Hct WNL, by ….
  • 33. Interventions for FVD - Hypovolemia  Prevent further fluid loss  Oral rehydration therapy  IV therapy  Medications; antiemetics, antidiarrheals  Monitor CV, Resp, Renal, GI status  Monitor electrolytes – possible supplement rx  MONITOR WEIGHT and I & O
  • 34. Fluid Volume Excess FVE - Hypervolemia  Fluid overload is an excess of body fluid - overhydration  Excess fluid volume in the intravascular area-hypervolemia  Excess fluid volume in interstitial spaces edema
  • 35. Fluid Volume Excess  Causes:  Increased Na/H2O retention  Excessive intake of Na (PO or IV)  Excessive intake of H2O ( PO or IV) (Water intoxication)  Syndrome of inappropriate antidiuretic hormone (SIADH)  Renal failure, congestive heart failure
  • 36. Assessment FVE - Hypervolemia CV: Elevated pulse; bounding, elevated BP, distended neck & hand veins, ventricular gallop (S3) Hyponatremia Resp: Dyspnea, Moist Crackles,Tachypnea Integumentary: Periorbital edema Pitting or Non-pitting edema GI: Increased motility Stomach cramps Nausea & Vomiting Renal: Weight gain Decreased spec grav of urine Neuromuscular: Altered LOC, headache, skeletal muscle twitching
  • 37. Nursing Diagnosis - FVE Fluid volume excess R/T excessive H20 intake confusion, headache, muscle twitching, abdominal cramps, elevated BP and HR, hyponatremia.
  • 38. Planning - FVE  Client will demonstrate fluid balance by balanced I & O measurements, Serum Na WNL, etc. by ….
  • 39. Interventions FVE - Hypervolemia  Restore normal fluid balance, prevent further overload  Drug therapy; diuretics  Diet therapy; decrease Na & fluids  Monitor intake and output (I & O)  Monitor weights  Monitor electrolytes  Monitor CV, Resp, Renal systems
  • 40. Electrolytes  Work with fluids to keep the body healthy and in balance  They are solutes that are found in various concentrations and measured in terms of milliequivalent (mEq) units  Can be negatively charged (anions) or positively charged (cations)  For homeostasis body needs: Total body ANIONS = Total body CATIONS
  • 41. Electrolytes Cations Positively charged  Sodium Na+  Potassium K+  Calcium Ca++  Magnesium Mg++ Anions Negatively charged  Chloride Cl-  Phosphate PO4-  Bicarbonate HCO3-
  • 42. Electrolyte Functions  Regulate water distribution  Muscle contraction  Nerve impulse transmission  Blood clotting  Regulate enzyme reactions (ATP)  Regulate acid-base balance
  • 43. Sodium Na+  135-145mEq/L  Major Cation  Chief electrolyte of the ECF  Regulates volume of body fluids  Needed for nerve impulse & muscle fiber transmission (Na/K pump)  Regulated by kidneys/ hormones
  • 44. Hmmm… Hyper and Hypo Natremia are the most common electrolyte disturbances. Why do you think that is? It is most abundant in the EXTRACELLULAR FLUID and therefore more prone to fluctuation.
  • 45. Hyponatremia  Serum Na+ <135mEq/L  Results from excess of water or loss of Na+  Water shifts from ECF into cells  S/S: abd cramps, confusion, N/V, H/A, pitting edema over sternum  Tx: Diet/IV therapy/fluid restrictions
  • 46. Lets think about … Hyponatremia  What are some medical conditions that may cause a dilutional hyponatremia? CHF Renal Failure SIADH ( Cancer, pituitary trauma ) Addisons Disease ( hypoaldosteronism & Na loss )  What are some conditions that might cause actual loss of sodium from the body? GI losses – nasogastric suctioning, vomiting, diarrhea Certain diuretic therapies  Permanent neurological damage can occur when serum Na levels fall below 110 mEq/L. Why? Hypotonic environment swells cells, increasing ICP – brain damage
  • 47. Hypernatremia  Serum Na+> 145mEq/L  Results from Na+ gained in excess of H2O OR Water is lost in excess of Na+  Water shifts from cells to ECF  S/S: thirst, dry mucous membranes & lips, oliguria, increased temp & pulse,flushed skin,confusion  Tx: IV therapy/diet
  • 48. Let’s think about…. Hypernatremia  What are some medical conditions that may cause elevated serum Na? Renal failure Diabetes Insipidus Diabetes Mellitus ( hyperglycemic dehydration) Cushings syndrome (hyperaldosteronism)  What are some other patient populations at risk for hypernatremia? Elderly ( decreased thirst mechanism ) Patient’s receiving: -tube feedings -corticosteroid drugs -certain diuretic therapies  Seizures, coma, death my result if hypernatremia is left untreated. Why?
  • 49. Potassium K+  3.5-5.0 mEq/L  Chief electrolyte of ICF  Major mineral in all cellular fluids  Aids in muscle contraction, nerve & electrical impulse conduction, regulates enzyme activity, regulates IC H20 content, assists in acid-base balance  Regulated by kidneys/ hormones  Inversely proportional to Na
  • 50. Hypokalemia  Serum level < 3.5mEq/L  Results from decreased intake, loss via GI/Renal & potassium depleting diuretics  Life threatening-all body systems affected  S/S muscle weakness & leg cramps, decreased GI motility, cardiac arrhythmias  Tx: diet/supplements/IV therapy
  • 51. Lets think about … Hypokalemia  What are some medical conditions that may cause a hypokalemia? Renal Disease / CHF (dilutional) Metabolic Alkalosis Cushings Disease ( Na retention leads to K loss )  What are some conditions that might cause actual loss of potassium from the body? GI losses – nasogastric suctioning, vomiting, diarrhea Certain diuretic therapies Inadequate intake – ( body cannot conserve K, need PO intake)  Cardiac arrest may occur when serum K levels fall below 2.5 mEq/L. Why? Increased cardiac muscle irritability leads to PACs and PVCs, then AF
  • 52. Hyperkalemia  Serum level >5 mEq/L  Results from excessive intake, trauma, crush injuries, burns, renal failure  S/S muscle weakness, cardiac changes, N/V, parathesias of face/fingers/tongue  Tx:diet/meds/IV therapy/ possible dialysis
  • 53. Lets think about … Hyperkalemia  What are some medical conditions that may cause hyperkalemia? Renal Disease=most common cause Burns and other major tissue trauma Metabolic Acidosis Addison’s Disease ( Na loss leads to K retention )  What are some conditions that might cause potassium levels to rise in the body? Certain diuretic therapies Excessive intake – ( inappropriate supplements)  Cardiac arrest may occur when serum K levels rise above mEq/L. Why? Decreased electrical impulse conduction leads to bradycardia and eventual asystole.
  • 54. Calcium Ca++  4.5-5.5mEq/L  Most abundant in body but: 99% in teeth and bones  Needed for nerve transmission, vitamin B12 absorption, muscle contraction & blood clotting  Inverse relationship with Phosphorus  Vitamin D needed for Ca absorption
  • 55. Hypocalcemia  Serum Ca < 4.3mEq/L  Results from low intake, loop diuretics, parathyroid disorders, renal failure  S/S osteomalacia, EKG changes, numbness/tingling in fingers, muscle cramps / tetany, seizures, Chovstek Sign & Trousseau Sign  Tx: diet/IV therapy
  • 56. Chovstek Trousseau
  • 57. Lets think about … Hypocalcemia  What are some medical conditions that may cause hypocalcemia? Hypoparathyroidism (low PTH levels = decreased release of Ca from bones) S/P thryoid surgery ( low Calcitonin = decreased release of Ca from bones) Acute pancreatitis Crohns Disease Hyperphosphatemia ( ESRF)  What are some other conditions that might cause low Ca? GI losses – nasogastric suctioning, vomiting, diarrhea Long term immobilization Lactose intolerance  If hypocalcemia is prolonged, the body will utilize stored Ca from bones. What complication might arise?
  • 58. Hypercalcemia  Serum Ca > 5.3mEq/L  Results from hyperparathyroidism, some cancers, prolonged immobilization  S/S muscle weakness, renal calculi, fatigue, altered LOC, decreased GI motility, cardiac changes  Tx: medication/ IV therapy
  • 59. Lets think about … Hypercalcemia  What are some medical conditions that may cause hypercalcemia? Hyperparathyroidism (high PTH levels = increased release of Ca from bones) Paget’s Disease Some Cancers – Multiple Myleoma Chronic Alcoholism ( with low serum phosphorus )  What are some other conditions that might cause low Ca? Excessive intake of Ca OR Vitamin D Excessive intake of OTC antacids If hypercalcemia is uncorrected, AV block and cardiac arrest may occur.
  • 60. Magnesium Mg2+  1.5-2.5mEq/L  Most located within ICF  Needed for activating enzymes, electrical activity, metabolism of carbs/proteins, DNA synthesis  Regulated by intestinal absorption and kidney
  • 61. Hypomagnesemia  Serum < 1.5mEq/L  Results from decreased intake, prolonged NPO status, chronic alcoholism & nasogastric suctioning  S/S: muscle weakness, cardiac changes, mental changes, hyperactive reflexes & other hypocalcemia S/S.  Tx: replacement IV therapy restore normal Ca levels ( Mg mimics Ca) seizure precautions
  • 62. Hypomagnesemia  Common in critically ill patients  Associated with high mortality rates  Increases cardiac irritability and ventricular dysrhythmias - especially in patients with recent MI  Maintenance of adequate serum Mg has been shown to reduce mortality rates post MI
  • 63. Hypermagnesemia  Serum>2.5mEq/L  Results from renal failure, increased intake  S/S: flushing, lethargy, cardiac changes (decreased HR),decreased resp, loss of deep tendon reflexes  Tx: restrict intake diuretic rx
  • 64. Chloride Cl-  95-105mEq/L  Most abundant anion in ECF  Combines with Na to form salts  Maintains water balance, acid-base balance, aids in digestion (hydrochoric acid) & osmotic pressure (with Na and H20)  Regulated by kidneys  Follows Sodium (Na)
  • 65. Hypochloremia  Serum level 96mEq/L  Results from prolonged vomiting & suctioning  S/S metabolic alkalosis, nerve excitability, muscle cramps, twitching, hypoventilation, decreased BP if severe  Tx: diet/IV therapy
  • 66. Hyperchloremia  Serum level > 106mEq/L  Results from excessive intake or retention by kidneys – metabolic acidosis  S/S Arrhythmias, decreased cardiac output, muscle weakness, LOC changes, Kussmauls’s respirations  Tx: restore fluid & electrolyte balance
  • 67. Phosphate PO4-  2.5-4.5mg/dl  Needed for acid-base balance,neurological & muscle function, energy transfer ATP & affects metabolism of carbs/proteins/lipids, B vitamin synthesis  Found in the bones  Regulated by intake and kidneys  Inversely proportional to Calcium Therefore some regulation by PTH as well
  • 68. Hypophosphatemia  Serum level < 1.8mEq/L  Results from decreased intestinal absorption and increased excretion  S/S bone & muscle pain, mental changes, chest pain, resp. failure  Tx: Diet/ IV therapy
  • 69. Hyperphosphatemia  Serum level> 2.6mEq/L  Results from renal failure, low intake of calcium  S/S: neuromuscular changes (tetany), EKG changes, parathesia-fingertips/mouth  Tx: Diet; hypocalcemic interventions Medications: phosphate binding  The body can tolerate hyperphosphatemia fairly well BUT the accompanying hypocalcemia is a larger problem!
  • 70. Electrolyte homeostasis  This means to maintain balance… to control by balancing the dietary intake of electrolytes with the renal excretion and reabsorption of electrolytes
  • 71. Interventions for F/E balance  Assess patient carefully- note changes  Monitor I & O (Intake & Output)  Monitor weight changes  Monitor urine  Monitor vs  Monitor lab results and dx test  Maintain proper IV therapy
  • 72. Summary  Fluid compartments in the body must balance  Body systems regulate F&E balance  Assessment of body fluid is important to determine causes of imbalance  Interventions for imbalances are based on the cause