Alzheimer’s & Related Dementia

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A brief introduction to Alzheimer's disease and Dementia

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Alzheimer’s & Related Dementia

  1. 1. ALZHEIMER’S & RELATED DEMENTIA
  2. 2. ―While I am still able, I want to articulate and give voice to what it is like to walk in these shoes and let people know that given this diagnosis, we are capable of contributing to the world around us. Please listen to our voices – individually and collectively.‖ -Richard (who has early onset Alzheimer’s disease)
  3. 3. DEMENTIA  Dementia is the loss of cognitive functioning—thinking, remembering, and reasoning—to such an extent it interferes with a person’s daily life and activities. It is not a disease itself, but a group of symptoms that often accompanies a disease or condition (www​.nia.nih.g​ov/sites/d​efault/fil​es/Unr avel​ing_final).
  4. 4. EXAMPLES OF DISEASES THAT CAUSE DEMENTIA Alzheimer’s disease Vascular dementia Parkinson’s disease with dementia Pick’s disease Mixed dementia Dementia with Lewy bodies Frontotemporal dementia
  5. 5. OTHER CAUSES OF DEMENTIA Medication side effects Depression Vitamin B12 deficiency Chronic alcoholism Tumors or Infections of the Brain
  6. 6. ALZHEIMER’S DISEASE, A FORM OF DEMENTIA  Alzheimer's is the most common form of dementia, a general term for memory loss and other intellectual abilities serious enough to interfere with daily life. Alzheimer's disease accounts for 50 to 80 percent of dementia cases (www.alz.org).
  7. 7. ALZHEIMER’S DISEASE  Progressive brain disorder  Damages and eventually destroys brain cells  Leads to loss of memory, thinking and other brain functions  Not a part of normal aging  Starts slowly and gradually gets worse  Alzheimer's is fatal, and currently, there is no cure
  8. 8. ALZHEIMER’S BRAIN ABNORMALITIES Plaques, microscopic clumps of a protein called beta-amyloid peptide  Tangles, twisted microscopic strands of the protein tau (rhymes with "wow")  Loss of connections among brain cells responsible for memory, learning and communication. These connections, or synapses, transmit information from cell to cell.  Inflammation resulting from the brain's effort to fend off the lethal effects of the other changes under way  Eventual death of brain cells and severe tissue shrinkage 
  9. 9. PLAQUES & TANGLES
  10. 10. BRAIN CHANGES
  11. 11. THE BRAIN-A FEW HIGHLIGHTS Cerebral Cortex (outer layer)—processes sensory information from outside world  Frontal Lobe (front of brain)—thinking, organizing, planning and problem solving, memory, attention and movement  Parietal Lobe (behind frontal lobe)–perception and integration of stimuli from senses  Occipital Lobe (back of brain)–vision  Temporal Lobe–balance and coordination  Amygdala—processing and remembering strong memories (like fear)  Hippocampus—learning and short term memory. 
  12. 12. WHAT WE DO FOR PEOPLE WITH ALZHEIMER’S DISEASE AND OTHER FORMS OF DEMENTIA  Pharmacological (medication) Interventions (like Namenda & Aricept)  Non-pharmacological interventions  Provide support and education to family members  Advocate

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