Your SlideShare is downloading. ×
0
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Diabetes
Upcoming SlideShare
Loading in...5
×

Thanks for flagging this SlideShare!

Oops! An error has occurred.

×
Saving this for later? Get the SlideShare app to save on your phone or tablet. Read anywhere, anytime – even offline.
Text the download link to your phone
Standard text messaging rates apply

Diabetes

396

Published on

Relationship of Dentistry and Diabetes

Relationship of Dentistry and Diabetes

0 Comments
0 Likes
Statistics
Notes
  • Be the first to comment

  • Be the first to like this

No Downloads
Views
Total Views
396
On Slideshare
0
From Embeds
0
Number of Embeds
0
Actions
Shares
0
Downloads
12
Comments
0
Likes
0
Embeds 0
No embeds

Report content
Flagged as inappropriate Flag as inappropriate
Flag as inappropriate

Select your reason for flagging this presentation as inappropriate.

Cancel
No notes for slide

Transcript

  • 1. DIABETES SUPPORT GROUP DAVID L. BURNS D.D.S. JANUARY 7, 2004
  • 2. M a y a C lin ic A n In ter view W ith R o b ert R izz a, M .D . W H S : W h a t is diab etes ? bu t it's n ot r ea lly n ecessa r y; th e th era p y is D r. R izza : D ia b ete s is a d is ea s e in w h ich you r b lood sa m e. 8 s u ga r (glu cos e ) con ce n tra tion is high e r tha n n orm a l. W H S : C a n dia be te s be p re ve n te d ? T h e s e high le ve ls ca n occu r a n y- tim e du rin g th e d a y. A h igh le vel e arly in the m ornin g is re ferre d to as a D r. R izz a: Y es. G en era lly, you ca n pr even h igh fa stin g glu - cos e . W he n it's h igh a fte r you e at a typ e 2 d ia betes if you sta y lea n an d fit. T h m e a l, it's re fe rre d to a s a high p os tpran d ia l glu cos e . . you r bod y h a s th e a bility to p r od u ce en ou in su lin for you r n eed s. .W ith typ e 1 , it's m com p lica ted . T h is is a disea se in w h ich yo W H S : W h a t's the diffe re n ce b e tw e e n typ e 1 a n d d ev elop a n tibod ies or som e im - m un e ty pe 2 dia b ete s? a bn orm alities ag a in st th e beta cells in you D r. R izz a: T h ey'r e d iffer en t d isea se pr ocesse s. In p an cr ea s. B u t in ter estin g ly, ever ybod y w h typ e 1 (for m erly ca lled ju ven ile or in su lin - th ese im m un e a bn orm alities d oesn 't n eces d ep en d en t d ia betes) you r bod y d estr o ys a ll th e d ev elop t yp e 1 d ia betes. F or ex a m - ple, if in su lin -secr etin g cells (beta cells) in you r h a ve an id en tical tw in w h o h a s p an cr ea s. T h is is believ ed to be a d isord er of you r typ e 1 d ia betes, you on l y h a ve a p pr ox im a in tern al im m un e system . .In typ e 2 (for - m er ly p er cen t ch an ce of g ettin g th e d isea se, too. ca lled a d u lt-on set or n on in su lin -d ep en - d en t tested th e a un ts, un cles, br oth er s, sis- ter s, d ia betes) you r p an cr ea s m ak es in su lin , bu t n ot m oth er s an d fa th er s of p eop le w ith typ e
  • 3. A n In ter view W ith R o b ert R izz a, M .D . W H S : W h a t is diab etes ? D r. R izza : D ia b ete s is a d is ea s e in w h ich you r b lood s u ga r (glu cos e ) con ce n tra tion is high e r tha n n orm a l. T h e s e high le ve ls ca n occu r a n y- tim e du rin g th e d a y. A h igh le vel e arly in the m ornin g is re ferre d to as a h igh fa stin g glu - cos e . W he n it's h igh a fte r you e at a m e a l, it's re fe rre d to a s a high p os tpran d ia l glu cos e . .
  • 4. W H S : W h a t's the diffe re n ce b e tw e e n typ e 1 a n d ty pe 2 dia b ete s? D r. R izz a: T h ey'r e d iffer en t d isea se pr ocesse s. In typ e 1 (for m erly ca lled ju ven ile or in su lin - d ep en d en t d ia betes) you r bod y d estr o ys a ll th e in su lin -secr etin g cells (beta cells) in you r p an cr ea s. T h is is believ ed to be a d isord er of you r in tern al im m un e system . .In typ e 2 (for - m er ly ca lled a d u lt-on set or n on in su lin -d ep en - d en t d ia betes) you r p an cr ea s m ak es in su lin , bu t n ot en ou g h for you r bod y's n eed s. T h at can be be ca u se you r bod y n eed s a lot of in sulin , w h ich is r eferr ed to a s in sulin r esis- tan ce, or it cou ld be be ca u se you r bod y h a s gr ea t d ifficu lty s ecr etin g in su lin . T h er e's a lw a ys a ba lan ce betw een h ow m u ch in su lin you r bod y m a k es ver su s h ow m u ch you r
  • 5. b od y n eed s. F or ex am ple, you r bod y m a k es a cer tain a m oun t of in sulin . B u t if you 'r e over - w eig h t or you 'r e sed en tar y, you r bod y n eed s m or e. Y ou r blo od su g ar g oes u p beca u se you ca n 't secr ete a s m u ch in sulin a s you n eed . O n th e oth er h an d, you c ou ld h a ve a g en etic a bn or m a lity in th e w a y you secr ete in su lin , so even th ou gh you 'r e lean an d fit, you still m a y n ot h a ve q u ite en ou g h , an d you r blood su g ar g oes u p . T h is is w h y you m a y see a th in old er a d u lt w ith typ e 2 d ia betes or a ver y h ea v y you n g p er son w ith typ e 2 d ia betes. .
  • 6. W H S : W h ic h ty pe is m o st like ly to a ffec t a d ult wom en? D r. R izz a: M ost a d ult w om en w h o h a ve d ia- bete s h a ve typ e 2 . T h at's n ot to sa y th at you ca n 't d ev elop t yp e 1 a s an a d ult. In fa ct, a bou t 1 0 p er cen t to 15 p er cen t of a d u lts w h o d evel- op d ia betes h a ve typ e 1 . C erta in tests can be u sed to h elp d eter m in e w h ich typ e you h a ve,
  • 7. WHS: Can diabetes be prevented? Dr. Rizza: Yes. Generally, you can prevent type 2 diabetes if you stay lean and fit. That way your body has the ability to produce enough insulin for your needs. With type 1, it’s more complicated. This is a disease in which you develop antibodies or some immune abnormalities against the beta cells in your pancreas. But interesting- ly, everybody who has these immune abnormalities doesn’t necessarily develop type 1 diabetes. For example, if you
  • 8. F or ex a m - ple, if you h a ve an id en tica l tw in w h o has typ e 1 d ia betes, you on l y h a ve a p pr ox im a tely a 5 0 p er cen t ch an ce of g ettin g th e d isea se, too. If you tested th e a un ts, un cles, br oth er s, sis- ter s, m oth er s an d fa th er s of p eop le w ith typ e 1 d ia betes, you 'd fin d th a t m an y h a ve an ti- bod ies a g ain st th eir beta cells, w h ich sa ys th is d estr u ctive p r ocess is g oin g on , yet th ey n ever g et dia betes. T h e pr oblem w e h a ve w ith typ e 1 d ia betes is th a t w e d on 't kn ow h ow to p r e- dict w h o is g oin g to g o on to d evel op th e dis- ea se an d w h o isn 't. .W e believe th a t som e oth er pr oces s is g oin g on . B esid es a g en etic pr ed isp osition , p erh ap s an en vir on m en tal tr igg er is n ecessa r y or som e oth er in itia tin g I fa ct orth at carries th e d isea se pr ocess for w a r d , r esu ltin g in dia betes. .
  • 9. W H S : W h a t is p re -dia be te s? D r. R izz a: Pr e-dia betes is a term th at im p lies th at you h a ve cer tain fa ctor s th a t p la ce you a t h igh risk o f p r ogr essin g to d ia betes. F or in stan ce, a n or m a l fa stin g blood su g ar is 8 0 to 1 00 . Y ou h a ve d ia betes if you r fa stin g blood su g a r is m or e th an 1 2 6 . B u t if you h a ve a blood su g ar th at's betw een 1 0 0 an d 1 2 5, you d on 't h a ve d ia betes yet, bu t clea rly you r blo od su g a r is alr ea d y a bn orm al. T h is is w h en you r d octor m a y sa y you 'r e pr e-d ia betic. H a vin g a blo od su g a r in th is pr e-d ia betes I ran g e p uts you a t a ver y h igh risk of p r ogr ess- in g to d ia betes u n less you d o som eth in g a bou t it, su ch a s lose w eig h t an d ex er cise. Y ou
  • 10. m a y n ot h a ve fa lle n off th e sid e of a cliff, bu t you 're ge ttin g p re tty clos e to th e e d ge . . W H S : W h o sh o u ld b e sc re e ne d fo r d ia be te s? D r. R izz a: T h e A m erican D ia betes A ssocia tion (A D A ) sa ys th a t ever yon e, par ticu larly th ose w h o ar e over w eig h t, sh ou ld be scr een ed for dia betes ev er y th r ee yea r s beg in n in g a t a g e 4 5 . T estin g sh ou ld be m or e fr eq u en t for p eop le w h o ar e o ver w eig h t w h o a lso h a ve oth er risk fa ctor s su ch a s a fa m ily h istor y o f d ia betes, a sed en tar y lifest yle, h igh blood p r essur e, an d h igh ch olester ol an d tr iglycer id e levels. 8
  • 11. W H S : W h a t te sts a re do n e to sc re e n fo r dia b ete s? D r. R izz a: T w o tests ar e m ost often d on e. O n e is a sim p le blood test ca lled a fa stin g blood su g ar . T h is test is ea sier an d fa ster to p er for m . If you r r esu lt is gr eater th an 1 2 6, th en it in di- ca tes d ia betes. T h e oth er test is ca lled a tw o- h ou r g lu cose toler an ce test. In th a t test you 'r e g iven a sw eet liq u id to dr in k an d th en tw o h ou r s later a blo od su g a r test is d on e. If th e r esu lt is gr eater th an 2 0 0, th en you h a ve d ia - betes. T h e A D A r ecom m en d s a fa stin g blood su g a r test fir st, bu t if you 'r e a t h igh risk of d ia betes -or th e r esu lts of you r fa stin g test ar e a bn orm al -you r d octor m a y con sid er d oin g a tw o-h ou r glu cose toler an ce test. 8
  • 12. W H S : W h a t a re the tre atm e nts fo r dia be te s? D r. R izz a: T r eatm en t for d ia betes is m ean t to r estor e th e ba lan ce bet w e en in su lin n eed an d in su lin a va ila bility. W e star t w ith lifest yle m od ifica tion -losin g w eig h t an d ex er cisin g. If you ca n a ccom p lish th ese, you ca n dram a ti- ca lly d ecr ea se h ow m u ch in su lin you r bod y r eq u ir es, so you m a y n eed to d o n oth in g m or e. O f cou r se, th is is ea sier sa id th an d on e. M an y p e op le n eed to ta k e m ed ica tion s for d ia betes. T h er e ar e th r ee m a jor cla sses of m ed - ica tion s -pills th a t m a k e you se cr ete m or e in su lin , p ills th a t m a k e th e liver pr od u ce less g lu cos e, an d p ills th a t m a k e in su lin w or k bet- ter. In su lin is u sed w h en p ills ar en 't en ou gh to k eep blo od su g ar in th e n orm al ran g e. .
  • 13. W H S : W h a t n ew tr ea tm en ts ar e o n th e h o rizo n ? D r. R izza : T h e re are a va riety o f n e w trea t- m e nts b ein g s tu died , s om e of th e m a t M a yo. A ll the on es m e n tion e d h e re a re in a d va n ce d sta ge s of tes tin g an d m a y b e rea d y w ithin the n e xt on e to thre e ye a rs. .O n e m e d icine com e s from a ga s trointe stin al h orm on e ca lle d G LP1,
  • 14. Ith e h or m on e th a t tells you r pan cr ea s th at food is com in g an d to g et r ea d y to secr ete in su lin . T h is m ed icin e is r em ark a ble beca u se it stim u - lates in su lin secr etion , it in h ibits glu ca g on - th e h orm on e th a t r aises blo od su g ar -an d it d ela ys th e ra te a t w h ich food em p ties fr om you r stom a ch , so th e m ed icin e can ea se in to you r blood str ea m . B u t w h a t's par ticu larly en cou r a g in g is th a t G L P l m a k es you secr ete in su lin on ly w h en you r blo od su g ar is h igh , so th e risk of g ettin g low blo od su g ar (h yp o- g lycem ia ) is n eg lig ible. .T h e pr oblem w ith
  • 15. th e G L P h orm on e is th a t it on ly la sts for a bou t a m in ute or tw o an d th en is ra p idly d eg r a d ed . B u t w h a t's bein g d evelop ed n ow a r e an a log s to th e h orm on e, in oth er w or d s, sim ilar su b- stan ces th a t ar en 't d egr a d ed so q u ick ly. .In oth er stu dies, a d r ug is bein g d evelop ed th a t in h ibits th e en z ym e th at d egra d es G L P . S o r ath er th an givin g you G L P , you w ou ld ta k e a pill to bo ost you r bod y's n a tura l G L P , g ivin g you th e sa m e effe ct o f stim ulatin g in su lin secr etion . .S tu dies ar e bein g d on e to fin d bet- ter w a ys o f d eliver in g in su lin th an b y g ivin g you r self in jection s ever y d a y. O n e sh ow in g pr om ise is in sulin th a t you in h ale. S om e d a ta sa y it w or k s fin e, bu t n ow th e q u estion is w h eth er it'll be sa fe for lon g -term u se. .
  • 16. W H S : W h a t o th e r im p o rtan t fac ts w o u ld y o u like o u r re a d e rs to k n o w a b o u t? D r. R izz a: Y ou h ear so m u ch a bou t all th e ba d h ealth con seq u en ces of d ia betes. A n d it's tr u e th at d ia betes is th e n um ber on e cau se of k id - n ey d isea se, blin dn ess, am p uta tion s, h eart d is- ea se an d m or e. H o w e v er , th ese con seq u en ces h a p p en on ly w h en d ia betes is un tr ea ted . If you tr eat you r d ia betes p r op erly, k eep you r blood su g ar n or m a l, k eep you r blood p r essur e n orm al an d k eep you r blo od lip id s n orm al, you 'r e g oin g to be fin e. .
  • 17. W H S : W h a t's y o u r h o pe fo r the .futu re o f dia b ete s c a re ? D r. R izz a: O f cou r se, th e ultim a te g oa l is to cur e d ia betes. U n til th en , m y h op e for th e fu tur e is th a t ev er yon e w ill be a ble to h a ve a ccess to th e ver y be st car e for th eir dia betes. D ia betes r eq u ir es a p ar tn er sh ip betw e en p eo- p le, th eir ph ysicia n s an d oth er h ealth car e pr ovid er s. W e h a ve m eth od s a va ila ble n ow th at can n orm a liz e a lm ost ev er yb od y's blo od su g a r s. B u t you n eed to w or k tog eth er a s a tea m to m a k e th e m ost of th ese m eth od s. .
  • 18. C en ter piece E nam el D e n tin P u lp O ra l h e a lth c a re is im p o rta n t th ro u g h o u t a ll th e s ta g e s o f y o u r life . H o w e v e 1 ; s e v e ra l c o n - d itio n s a s s o c ia te d w ith a g in g c a n le a d to m o u th C ro w n a n d g u m d is e a s e . In a d d itio n , d e n ta l d is e a s e is n o w c o n s id e re d a c o n trib u tin g fa c to r fo r s o m e c a s e s o f h e a rt d is e a s e , p n e u m o n ia , s tro ke a n d u n co n tro lle d d ia b e te s. Root A cc or d in g to th e S ur g eon H ea lth y teeth an d g um s w ou ld n 't r em ain G en era l's r ep ort "O ra l H ea lth in h ealth y for lon g w ith ou t saliva . It's secr eted fr om A m erica ,1 I th e g lan d s th r ou gh ou t you r m ou th . It h elp s clean you r h ealth of you r m ou th r eflects you r m ou th an d teeth , aid s in sw a llow - in g, an d g en era l h ealth an d w ell- con ta in s en z ym es th a t aid d ig estion an d h elp Bone C e m e n tum N e rv e a n d bein g . H o w e ver , ora l h ealth is often con tr ol in fection . b lo o d s up p ly o ver look ed an d un d er em ph a siz ed. A lm ost ever yon e is su scep tible to g u m d is- ea se an d tooth d eca y. H o w e ver , som e fa ctor s m a y N a tur a l look of tee th a n d g um s in cr ea se you r risk . T h ese in clu d e la ck of Y ou r m ou th is p erh ap s th e m ost m u ltifun c- tion a l flu or id ated w a ter, p oor d iet, fr eq u en t sn a ck in g on T h e s u r fa c e o f a sw eets, h er ed ity an d toba cc o u se. p ar t of you r bod y. O f you r m ou th 's m an y fu n ction s, th e m ost obvi ou s ar e bitin g , ch ew in g h e a lth y c r o w n is P la y in g h ost to b a c te ria an d sw a llow in g fo od . T h e m ost im p or tan t tools in th is pr oces s ar e you r teeth . Y ou r m ou th , lik e m an y oth er p arts of you r bod y,
  • 19. A c c o r d in g to th e S u r g e o n G e n e ra l's r e p o rt " O ra l H e a lth in A m e ric a ” I th e ,1 h e a lth o f y o u r m o u th r e fle c ts y o u r g e n e ra l h e a lth a n d w e ll- b e in g . H o w e v e r , o ra l h e a lth is o fte n o v e r lo o k e d a n d u n d er e m p h a s iz e d .
  • 20. N a tur a l look of tee th a n d g um s Y ou r m ou th is p erh ap s th e m ost m u ltifun c- tion a l p ar t of you r bod y. O f you r m ou th 's m an y fu n ction s, th e m ost obvi ou s ar e bitin g , ch ew in g an d sw a llow in g fo od . T h e m ost im p or tan t tools in th is pr oces s ar e you r teeth . E a ch of you r teeth -m ost p eop le d evel op 3 2 of th em -can be d ivid ed in to tw o p ar ts. T h e visible p or tion is th e cr ow n . T h e p or tion h id d en belo w th e g um lin e is th e r oot. T h e sur - fa ce o f a h ealth y cr ow n is cover ed w ith en a m - el, a th in la yer of ca lcified m aterial th at's th e h ar d est su bstan ce in th e bod y. T h e ou ter sur- fa ce of th e r oot is cover ed b y cem en tu m . T h e p eriod on tal liga m en t, a con n ective tissu e, bin d s r oot cem en tum to th e b on e th a t for m s th e tooth sock et.
  • 21. B en ea th th e en am el an d cem en tum lies th e b od y of th e in n er tooth (d en tin ). D en tin is softer th an en am el an d cem en tum , bu t h ard er th an bon e. It surr oun d s th e h ollow tooth cor e (ca lled th e p ulp ca n a l or r oot ca n a l), an ar ea w h er e n er ve en d in g s an d blood ves sels sen si- tiz e an d n our ish th e tooth . N er ves a n d vessels feed in to th e p ulp can al th r ou g h an op en in g at th e r oot tip. F irm , pin k g um s (gin g iva ) surr oun d a h ea lth y tooth . A t th e ed g e of you r gu m lin e, you r g um tissu e fold s ba ck u n d ern eath itself. T h is cr eates ar ou n d ea ch tooth a sn ug gr oov e ca lled th e g in g iva l su lcu s.
  • 22. H ea lth y teeth an d g um s w ou ld n 't r em ain h ealth y for lon g w ith ou t saliva . It's secr eted fr om g lan d s th r ou gh ou t you r m ou th . It h elp s clean you r m ou th an d teeth , aid s in sw a llow - in g, an d con ta in s en z ym es th a t aid d ig estion an d h elp con tr ol in fection . A lm ost ever yon e is su scep tible to g u m d is- ea se an d tooth d eca y. H o w e ver , som e fa ctor s m a y in cr ea se you r risk . T h ese in clu d e la ck of flu or id ated w a ter, p oor d iet, fr eq u en t sn a ck in g on sw eets, h er ed ity an d toba cc o u se.
  • 23. P la y in g h ost to b a c te ria Y ou r m ou th , lik e m an y oth er p arts of you r bod y, is h ost to ba cteria. T h ese ba cter ia con - ver t som e o f th e su g ar s an d car boh yd r a tes you ea t in to a cid . T h e ba cter ia an d a cid s th ey for m becom e p a rt of th e stick y d ep osit - ca lled d en ta l p la q u e -th at clin g s to th e sur - fa ce o f you r teeth . In ad d ition to ba cteria, p la q u e is com p osed o f sa liva an d fo od p ar ticles. O ver tim e, p la q u e can h ar d en an d for m a d ifficu lt-to-r em ove su bsta n ce ca lled tartar { ca lcu lu s).
  • 24. D ep en d in g on w h er e p la q u e an d tartar for m , th e y m a y ca u se: T o o th d ec a y (c a rie s) -D eca y is a ba cterial dis- ea se o f teeth an d th e pr im ar y ca u se of to oth loss. It is th e r esu lt of th r ee in ter a ctin g fa ctor s: ba cteria g r ow th , dietar y su g ar an d a vu ln er a - ble tooth su r fa ce. T h e d eca y-p r od u cin g a cid th a t for m s in p la q u e a tta ck s th e m in er als in th e tooth 's ou ter en am el sur fa ce. T h e er osion ca u sed b y th e p la q u e lea d s to tin y op en in g s { ca vities) in th e en a m el, w h ich you m a y n ot n otice in itially. T h e fir st sign of d eca y m a y be a sen sa tion of p a in w h en you ea t som eth in g sw eet, ver y c old or ver y h ot. O n ce th e en am el is p en etr ated , th e un d er lyin g softer d en tin
  • 25. D ep en d in g on w h er e p la q u e an d tartar for m , th e y m a y ca u se: T o o th d ec a y (c a rie s) -D eca y is a ba cterial dis- ea se o f teeth an d th e pr im ar y ca u se of to oth loss. It is th e r esu lt of th r ee in ter a ctin g fa ctor s: ba cteria g r ow th , dietar y su g ar an d a vu ln er a - ble tooth su r fa ce. T h e d eca y-p r od u cin g a cid th a t for m s in p la q u e a tta ck s th e m in er als in th e tooth 's ou ter en am el sur fa ce. T h e er osion ca u sed b y th e p la q u e lea d s to tin y op en in g s { ca vities) in th e en a m el, w h ich you m a y n ot n otice in itially. T h e fir st sign of d eca y m a y be a sen sa tion of p a in w h en you ea t som eth in g sw eet, ver y c old or ver y h ot. O n ce th e en am el is p en etr ated , th e un d er lyin g softer d en tin
  • 26. G in g iv itis -T h is m ild , ear ly for m of g u m dis- ea se ca n ca u se h ea lth y, p in k g um s to bec om e r ed , sw ollen , ten d er an d pr on e to bleed in g. T h e sw ellin g an d ten d ern ess a ssocia ted w ith g in g ivitis is ca u sed by you r im m un e system 's in fla m m ator y r esp on se to p la q u e or tartar bu ild up a lon g you r gu m lin e. G in g ivitis often is p ain less an d g oes u n n oticed . If un ch eck ed , g in g ivitis can lead to p eriod on titis, a m or e seriou s d isea se.
  • 27. P e rio d o n titis -T h is is an a d van ced sta g e of g u m d isea se th at m a y d e vel op w h en plaq u e an d tartar ex ten d ben ea th you r g um lin e. G ra d ua lly, you r g u m s w ith dra w fr om ar ou n d you r teeth . P ock ets o f in fection can for m in th is d ar k, a irless r eg ion an d d estr oy tissu e an d you r tooth sock ets (a lveola r bon e). L eft un tr eated , in volv ed teeth even tua lly lo osen an d fa ll ou t. C h r on ic p eriod on tal disea se is th e lead in g ca u se of tooth loss in old er ad u lts.
  • 28. R isk fa c tor s w ith a g e, g e n d er S om e or a l con d ition s ar e un iq u e to w om en at sp ecifi c p h a ses of th eir lives d u e to sev er a l fa c- tor s, in clud in g h orm on es. F or exa m ple, d ur in g m en op a u se, you m a y n otice d iscom for t in you r m ou th , in clud in g dr y m ou th an d bu rn - in g sen sa tion s in th e gu m tissu e (bu rn in g m ou th s yn d r om e). Y ou m a y a lso n otice alter - ation s in ta stes, su ch a s sa lty, p ep p er y or sou r .
  • 29. . C er ta in disea ses th at a ffe ct th e ora l ca v- ity a lso ar e m or e com m on in w om en . A n d, th e old er you ar e, th e gr eater you r risk of d evel- op in g d isea ses o f th e m ou th . A m on g th e fa c- tor s th a t m a y a ffe ct you r or a l h ealth ar e:
  • 30. D ia b ete s -T h is con dition p u ts you a t gr eater risk o f d ev elop in g p eriod on tal disea se an d oth er in fection s. It in cr ea ses even m or e if you r dia betes is p oor ly c on tr olled . T h at's beca u se d ia betes m a y w ea k en th e r esistan ce of you r g u m tissu e to ba cter ial in fection an d pla qu e. In ad d ition , or al in fection m a k es blood g lu - cose levels h ar d er to con tr ol.
  • 31. O ste o p o ro sis -R esea r ch er s su sp ect a lin k be- tw e en th e loss of b on e m in er al d en sity w ith oste op or osis an d p er iod on ta l d isea se. L osin g d en sity in th e bon es th a t h old you r teeth in pla ce m a y h elp sp eed th e d eg en era tive p r o- cess of p er iod on ta l d isea se. O steop or osis a lso m a y m a k e a g ood d en tur e fit d ifficu lt.
  • 32. M e d ic atio n s -S om e dr u g s -su ch a s an tid e- p r essan ts an d certain blo od pr essu r e dr ug s - can lea d to dr y m ou th , w h ich in cr ea ses you r risk of g u m pr oblem s an d tooth loss.
  • 33. W e a r a n d tea r -A s you g et old er , you m a y n otice th at you r m ou th feels dr ier an d you r g um s h a ve p u lled ba ck (r eced ed } . Y ou r teeth m a y d a rk en sligh tly a n d bec om e m or e brittle an d br ea ka ble.
  • 34. M ak in g th e e ffor t A d va n ces in d en tal tech n olog y -su ch a s g en etic testin g for or al disea se or d eca y r isk an d th e a bility to in itia te th e gr ow th of n ew teeth , bon e an d tissu e -m a y som ed a y p la ya p r om in en t r ole in d en tistr y. H o w e ver , da ily or a l h yg ien e com bin ed w ith r eg u lar d en ta l visits -a sk you r d en tist h ow o ften -is still th e best w a y to in cr ea se you r ch an ces of a h ea lth y m ou th . C arin g for you r m ou th an d g u m s m a y h elp to r eflect m or e th an a h ealth y sm ile. It m a y h elp to r eflect a h ealth ier you . 8
  • 35. 60y ear o ld me morial pre sentat ion Car iology & Per iodont ology Pr es en ted b y K. Ta mak i 2003.03.12 . Academy of Template Research Department of Dental & Oral Surgery Wakayama Medical University
  • 36. CARIES
  • 37. CAVITY/ ABSCESS
  • 38. STREPTOCOCCUS MUTANS
  • 39. CAVITIES AND PERIODONTAL DISEASE
  • 40. RADICULAR GRANULOMA
  • 41. RADICULAR CYST
  • 42. ALVEOLAR ABSCESS
  • 43. ABSCESS/ROOT CANAL THERAPY
  • 44. TREATMENT
  • 45. 外 傷 歯
  • 46. PERIODONTAL DISEASE
  • 47. Dental Plaque & Periodontal Disease
  • 48. ADVANCED PERIODONTAL D I S E A S E
  • 49. TARTAR/CALCULUS
  • 50. INFECTIOUS ORGANISMS
  • 51. INFECTION
  • 52. INFECTION

×